ArticleWorld journal of cardiology2009
Atherosclerosis, inflammation and Chlamydia pneumoniae.
Article in World journal of cardiology, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
8 citing papers in PubMed, 24 citations in OpenAlex.
- Bidirectional Interaction Between Chronic Kidney Disease andJournal of immunology research · 2025Article
- Article
- Productive and Penicillin-StressedFrontiers in cellular and infection microbiology · 2017Article
- Influence of intimal Chlamydophila pneumoniae persistence on cardiovascular complications after coronary intervention.Infection · 2015Article
- Article
- Is there any relationship between Chlamydophila pneumoniae and coronary atherosclerosis among Iranians?Nigerian medical journal : journal of the Nigeria Medical Association · 2013Article
- Chlamydia pneumoniae infection and cardiac risk factors in patients with myocardial infection.ARYA atherosclerosis · 2011Article
- Heart failure and chronic obstructive pulmonary disease: Two for tea or tea for two?World journal of cardiology · 2010Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Coronary heart disease is the single most common cause of illness and death in the developed world. Coronary atherosclerosis is by far the most frequent cause of ischemic heart disease, and plaque disruption with superimposed thrombosis is the main cause of the acute coronary syndromes of unstable angina, myocardial infarction, and sudden death. Atherosclerosis is the result of a complex interaction between blood elements, disturbed flow, and vessel wall abnormality, involving several pathological processes: inflammation, with increased endothelial permeability, endothelial activation, and monocyte recruitment; growth, with smooth muscle cell proliferation, migration, and matrix synthesis; degeneration, with lipid accumulation; necrosis, possibly related to the cytotoxic effect of oxidized lipid; calcification/ossification, which may represent an active rather than a dystrophic process; and thrombosis, with platelet recruitment and fibrin formation. In this review we discuss these processes and the possible pathological effects of Chlamydia infection and the ensuing phlogosis.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.