Evidence map›Paper›PMID 20972826›Full record

ReviewHeart failure reviews2011

The molecular fingerprint of cardiac dyssynchrony and cardiac resynchronization therapy.

Marc Vanderheyden, Chris Vrints, Jozef Bartunek

Abstract readReview
PubMed Publisher
In one paragraph

Review in Heart failure reviews, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
1.0field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 6 citations in OpenAlex.

  1. Cellular electrophysiological abnormalities in dyssynchronous hearts and during CRT.Journal of cardiovascular translational research · 2012
    Review
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Marc VanderheydenCardiovascular Center, Onze Lieve Vrouwziekenhuis, Aalst, Belgium. marc.vanderheyden@olvz-aalst.be
Chris Vrints
Jozef Bartunek
Onze Lieve Vrouwziekenhuis Hospital · BEUniversity of Antwerp · BE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

To date, cardiac resynchronization therapy remains the only treatment that enhances systolic function while improving long-term outcome and survival. Here, we review the molecular alterations associated with dyssynchronous heart failure and their reversibility induced by cardiac resynchronization therapy. We focus upon the molecular portrait of dyssynchronous heart failure and how cardiac resynchronization therapy influences electrophysiologic, metabolic and adrenergic pathways.

Indexed as

Cardiac Resynchronization TherapyAnimalsElectrophysiologyHeart FailureHumansProteomeReceptors, AdrenergicTreatment OutcomeVentricular Dysfunction, LeftProteomeReceptors, Adrenergic

Identifiers

PMID20972826
OpenAlexW2054433517

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.