ArticleJournal of theoretical biology2011
Cancer stem cell, niche and EGFR decide tumor development and treatment response: A bio-computational simulation study.
Article in Journal of theoretical biology, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
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Who cites it
17 citing papers in PubMed, 35 citations in OpenAlex.
- Nanoparticle-mediated cancer cell therapy: basic science to clinical applications.Cancer metastasis reviews · 2023Review
- Growth dynamics of breast cancer stem cells: effects of self-feedback and EMT mechanisms.Theory in biosciences = Theorie in den Biowissenschaften · 2022Article
- Optimization of Cancer Treatment in the Frequency Domain.The AAPS journal · 2019Article
- A function-blocking CD47 antibody suppresses stem cell and EGF signaling in triple-negative breast cancer.Oncotarget · 2016Article
- CD44(+)/CD24(-) breast cancer cells exhibit phenotypic reversion in three-dimensional self-assembling peptide RADA16 nanofiber scaffold.International journal of nanomedicine · 2015Article
- Article
- Dynamics between cancer cell subpopulations reveals a model coordinating with both hierarchical and stochastic concepts.PloS one · 2014Article
- A mathematical-biological joint effort to investigate the tumor-initiating ability of Cancer Stem Cells.PloS one · 2014Article
- Targeting tumor-infiltrating macrophages decreases tumor-initiating cells, relieves immunosuppression, and improves chemotherapeutic responses.Cancer research · 2013Article
- A 3D multiscale model of cancer stem cell in tumor development.BMC systems biology · 2013Article
- The relationship of cancer stem cells in urological cancers.Central European journal of urology · 2013Review
- Nonlinear growth kinetics of breast cancer stem cells: implications for cancer stem cell targeted therapy.Scientific reports · 2013Article
- Cancer stem cell theory: therapeutic implications for nanomedicine.International journal of nanomedicine · 2013Review
- N-cadherin impedes proliferation of the multiple myeloma cancer stem cells.American journal of blood research · 2013Article
- Application of circuit simulation method for differential modeling of TIM-2 iron uptake and metabolism in mouse kidney cells.Frontiers in physiology · 2013Article
- Multi-scale agent-based brain cancer modeling and prediction of TKI treatment response: incorporating EGFR signaling pathway and angiogenesis.BMC bioinformatics · 2012Article
- Lung cancer stem cell: new insights on experimental models and preclinical data.Journal of oncology · 2011Article
Corrections and comments
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Authors and funding
5 authors at 3 institutions in 2 countries.
Funding
Abstract
Recent research in cancer biology has suggested the hypothesis that tumors are initiated and driven by a small group of cancer stem cells (CSCs). Furthermore, cancer stem cell niches have been found to be essential in determining fates of CSCs, and several signaling pathways have been proven to play a crucial role in cellular behavior, which could be two important factors in cancer development. To better understand the progression, heterogeneity and treatment response of breast cancer, especially in the context of CSCs, we propose a mathematical model based on the cell compartment method. In this model, three compartments of cellular subpopulations are constructed: CSCs, progenitor cells (PCs), and terminal differentiated cells (TCs). Moreover, (1) the cancer stem cell niche is, considered by modeling its effect on division patterns (symmetric or asymmetric) of CSCs, and (2) the EGFR signaling pathway is integrated by modeling its role in cell proliferation, apoptosis. Our simulation results indicate that (1) a higher probability for symmetric division of CSC may result in a faster expansion of tumor population, and for a larger number of niches, the tumor grows at a slower rate, but the final tumor volume is larger; (2) higher EGFR expression correlates to tumors with larger volumes while a saturation function is observed, and (3) treatments that inhibit tyrosine kinase activity of EGFR may not only repress the tumor volume, but also decrease the CSCs percentages by shifting CSCs from symmetric divisions to asymmetric divisions. These findings suggest that therapies should be designed to effectively control or eliminate the symmetric division of CSCs and to reduce or destroy the CSC niches.
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.