ArticleVirology journal2010
What happened after the initial global spread of pandemic human influenza virus A (H1N1)? A population genetics approach.
Article in Virology journal, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
7 citing papers in PubMed.
- Molecular evolution and diversity of the norovirus RNA-dependent RNA polymerase.Scientific reports · 2026Article
- Genomic surveillance unfolds the SARS-CoV-2 transmission and divergence dynamics in Bangladesh.Frontiers in genetics · 2022Article
- Genetic diversity of SARS-CoV-2 in South America: demographic history and structuration signals.Archives of virology · 2021Article
- Association of clade-G SARS-CoV-2 viruses and age with increased mortality rates across 57 countries and India.Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases · 2021Article
- Anti-inflammatory effect of thalidomide on H1N1 influenza virus-induced pulmonary injury in mice.Inflammation · 2014Article
- Soluble RAGE as a severity marker in community acquired pneumonia associated sepsis.BMC infectious diseases · 2012Article
- Extensive geographical mixing of 2009 human H1N1 influenza A virus in a single university community.Journal of virology · 2011Article
Corrections and comments
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Authors and funding
9 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Viral population evolution dynamics of influenza A is crucial for surveillance and control. In this paper we analyzed viral genetic features during the recent pandemic caused by the new influenza human virus A H1N1, using a conventional population genetics approach based on 4689 hemagglutinin (HA) and neuraminidase (NA) sequences available in GenBank submitted between March and December of 2009. This analysis showed several relevant aspects: a) a scarce initial genetic variability within the viral isolates from some countries that increased along 2009 when influenza was dispersed around the world; b) a worldwide virus polarized behavior identified when comparing paired countries, low differentiation and high gene flow were found in some pairs and high differentiation and moderate or scarce gene flow in others, independently of their geographical closeness, c) lack of positive selection in HA and NA due to increase of the population size of virus variants, d) HA and NA variants spread in a few months all over the world being identified in the same countries in different months along 2009, and e) containment of viral variants in Mexico at the beginning of the outbreak, probably due to the control measures applied by the government.
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