Keeping the epidermal stem cell niche in shape.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

4 citing papers in PubMed.

1. Activation of G-Protein Coupled Receptor-Gαi Signaling Increases Keratinocyte Proliferation and Reduces Differentiation, Leading to Epidermal Hyperplasia.The Journal of investigative dermatology · 2020
   Article
2. JunB defines functional and structural integrity of the epidermo-pilosebaceous unit in the skin.Nature communications · 2018
   Article
3. Stem cell-based tissue engineering approaches for musculoskeletal regeneration.Current pharmaceutical design · 2013
   Review
4. Signaling circuitries controlling stem cell fate: to be or not to be.Current opinion in cell biology · 2011
   Review

Corrections and comments

• Comment on
  Actin and serum response factor transduce physical cues from the microenvironment to regulate epidermal stem cell fate decisions.2010

Authors and funding

2 authors.

Funding

Abstract