Evidence map›Paper›PMID 20652349›Full record

ReviewClinical and experimental nephrology2010

Searching for novel intercellular signal-transducing molecules in the kidney and their clinical application.

Kiyoshi Mori, Masashi Mukoyama, Kazuwa Nakao

Abstract readReview
PubMed Publisher
In one paragraph

Review in Clinical and experimental nephrology, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Trial
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Kiyoshi MoriDepartment of Medicine and Clinical Science, Kyoto University Graduate School of Medicine, 54 Shogoin Kawaharacho, Sakyo-ku, Kyoto 606-8507, Japan. keyem@kuhp.kyoto-u.ac.jp
Masashi Mukoyama
Kazuwa Nakao

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In this review, isolation and characterization of several kidney-derived molecules are described, namely carbonic anhydrase XIV, cysteine-rich protein 61, and kidney-liver-specific immunoglobulin-like protein. Features of neutrophil gelatinase-associated lipocalin (LCN2 or human neutrophil lipocalin) as a kidney differentiation inducer and renal injury biomarker and also as an iron-carrier protein are also summarized. Furthermore, the concepts of forest fire theory and the biology of siderophore-binding proteins are discussed.

Indexed as

Acute-Phase ProteinsAnimalsCarbonic AnhydrasesCysteine-Rich Protein 61HumansIntercellular Signaling Peptides and ProteinsKidneyKidney Failure, ChronicLipocalin-2LipocalinsMembrane ProteinsMiceOncogene ProteinsProto-Oncogene ProteinsSignal TransductionAcute-Phase ProteinsCarbonic Anhydrasescarbonic anhydrase XIVCysteine-Rich Protein 61Intercellular Signaling Peptides and ProteinsLCN2 protein, humanLcn2 protein, mouseLipocalin-2LipocalinsMembrane ProteinsOncogene ProteinsProto-Oncogene ProteinsTimd2 protein, mouse

Identifiers

PMID20652349

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.