A "rough" journey to the sarcoplasmic reticulum--implications of altered calsequestrin trafficking for cardiac arrhythmia.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

5 citing papers in PubMed.

1. An International Multicenter Evaluation of Inheritance Patterns, Arrhythmic Risks, and Underlying Mechanisms ofCirculation · 2020
   Trial
2. The structure of a calsequestrin filament reveals mechanisms of familial arrhythmia.Nature structural & molecular biology · 2020
   Article
3. The architecture and function of cardiac dyads.Biophysical reviews · 2020
   Review
4. New concepts of endoplasmic reticulum function in the heart: programmed to conserve.Journal of molecular and cellular cardiology · 2013
   Review
5. Calsequestrin 2 and arrhythmias.American journal of physiology. Heart and circulatory physiology · 2012
   Review

Corrections and comments

• Comment on
  Rough endoplasmic reticulum to junctional sarcoplasmic reticulum trafficking of calsequestrin in adult cardiomyocytes.2010

Authors and funding

1 author.

Funding

Abstract

PubMed holds no abstract for this paper.

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