Immunology. A guardian of T cell fate.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

12 citing papers in PubMed.

1. 14q32 rearrangements deregulating BCL11B mark a distinct subgroup of T-lymphoid and myeloid immature acute leukemia.Blood · 2021
   Trial
2. Bipartite DNA binding domain of transcription factor BCL11B binds clustered short DNA sequence motifs.bioRxiv : the preprint server for biology · 2026
   Article
3. Cxcr3 promotes protection from colorectal cancer liver metastasis by driving NK cell infiltration and plasticity.The Journal of clinical investigation · 2025
   Article
4. The role of transcription factors in shaping regulatory T cell identity.Nature reviews. Immunology · 2023
   Review
5. BCL11B depletion induces the development of highly cytotoxic innate T cells out of IL-15 stimulated peripheral blood αβ CD8+ T cells.Oncoimmunology · 2022
   Article
6. Distinct Notch1 and BCL11B requirements mediate human γδ/αβ T cell development.EMBO reports · 2020
   Article
7. Bcl11b prevents catastrophic autoimmunity by controlling multiple aspects of a regulatory T cell gene expression program.Science advances · 2019
   Article
8. Protein Kinase C-Mediated Phosphorylation of BCL11B at Serine 2 Negatively Regulates Its Interaction with NuRD Complexes during CD4+ T-Cell Activation.Molecular and cellular biology · 2016
   Article
9. Illegitimate V(D)J recombination-mediated deletions in Notch1 and Bcl11b are not sufficient for extensive clonal expansion and show minimal age or sex bias in frequency or junctional processing.Mutation research · 2014
   Article
10. Dll4-Notch signaling in Flt3-independent dendritic cell development and autoimmunity in mice.The Journal of experimental medicine · 2012
    Article
11. CD4-CD8 differentiation in the thymus: connecting circuits and building memories.Current opinion in immunology · 2012
    Review
12. Transcriptional control of natural killer cell differentiation and function.Cellular and molecular life sciences : CMLS · 2011
    Review

Corrections and comments

• Comment on
  Reprogramming of T cells to natural killer-like cells upon Bcl11b deletion.2010
• Comment on
  An early T cell lineage commitment checkpoint dependent on the transcription factor Bcl11b.2010
• Comment on
  An essential developmental checkpoint for production of the T cell lineage.2010

Authors and funding

1 author.

Funding

Abstract

PubMed holds no abstract for this paper.

Indexed as