Evidence map›Paper›PMID 20594413›Full record

ReviewRedox report : communications in free radical research2010

Oxygenomics in environmental stress.

H Sone, H Akanuma, T Fukuda

Open access · bronzeAbstract readReview
In one paragraph

Review in Redox report : communications in free radical research, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.4field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
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  9. Evaluation of systemic markers of inflammation in atomic-bomb survivors with special reference to radiation and age effects.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2012
    Article
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  14. Oxidative stress-mediated regulation of proteasome complexes.Molecular & cellular proteomics : MCP · 2011
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

H SoneNational Institute for Environmental Studies, 16-2 Onogawa, Tsukuba, Ibaraki, Japan. hsone@nies.go.jp
H Akanuma
T Fukuda
National Institute for Environmental Studies · JPTohoku University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Environmental stressors such as chemicals and physical agents induce various oxidative stresses and affect human health. To elucidate their underlying mechanisms, etiology and risk, analyses of gene expression signatures in environmental stress-induced human diseases, including neuronal disorders, cancer and diabetes, are crucially important. Recent studies have clarified oxidative stress-induced signaling pathways in human and experimental animals. These pathways are classifiable into several categories: reactive oxygen species (ROS) metabolism and antioxidant defenses, p53 pathway signaling, nitric oxide (NO) signaling pathway, hypoxia signaling, transforming growth factor (TGF)-beta bone morphogenetic protein (BMP) signaling, tumor necrosis factor (TNF) ligand-receptor signaling, and mitochondrial function. This review describes the gene expression signatures through which environmental stressors induce oxidative stress and regulate signal transduction pathways in rodent and human tissues.

Indexed as

GenomicsOxidative StressAnimalsAntioxidantsGene Expression ProfilingHumansHypoxiaMicroarray AnalysisReactive Oxygen SpeciesSignal TransductionTransforming Growth Factor betaAntioxidantsReactive Oxygen SpeciesTransforming Growth Factor beta

Identifiers

PMID20594413
PMCPMC7067327
OpenAlexW2009778778

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.