Evidence map›Paper›PMID 20453875›Full record

ReviewNature reviews. Microbiology2010

Carbon metabolism of intracellular bacterial pathogens and possible links to virulence.

Wolfgang Eisenreich, Thomas Dandekar, Jürgen Heesemann, Werner Goebel

Abstract readReview
PubMed Publisher
In one paragraph

Review in Nature reviews. Microbiology, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 197 papers.

0numbers the graph read from it
0cells of the map it votes in
197citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

197 citing papers in PubMed.

  1. Virulence · 2026
    Article
  2. Ability ofMicroorganisms · 2026
    Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. Review
  8. Article
  9. Gut microbes · 2025
    Article
  10. The veterinary quarterly · 2025
    Article
  11. Article
  12. eLife · 2025
    Article
  13. Article
  14. Article
  15. Cytoplasmic MgProceedings of the National Academy of Sciences of the United States of America · 2025
    Article
  16. Article
  17. Review
  18. Effect of vaccine diluents on the colonization ofVeterinary and animal science · 2024
    Article
  19. Article
  20. Automated Diagnosis and Phenotyping of Tuberculosis Using Serum Metabolic Fingerprints.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2024
    Article

137 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Wolfgang EisenreichLehrstuhl für Biochemie, Technische Universität München, D-85747 Garching, Germany.
Thomas Dandekar
Jürgen Heesemann
Werner Goebel

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

New technologies such as high-throughput methods and 13C-isotopologue-profiling analysis are beginning to provide us with insight into the in vivo metabolism of microorganisms, especially in the host cell compartments that are colonized by intracellular bacterial pathogens. In this Review, we discuss the recent progress made in determining the major carbon sources and metabolic pathways used by model intracellular bacterial pathogens that replicate either in the cytosol or in vacuoles of infected host cells. Furthermore, we highlight the possible links between intracellular carbon metabolism and the expression of virulence genes.

Indexed as

BacteriaBacterial ProteinsCarbonCytosolGenomic IslandsHost-Pathogen InteractionsListeria monocytogenesMycobacterium tuberculosisPeptide Termination FactorsSalmonella typhiShigella flexneriVacuolesVirulenceVirulence FactorsBacterial ProteinsCarbonPeptide Termination FactorsPrfA protein, Listeria monocytogenesVirulence Factors

Identifiers

PMID20453875

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.