ReviewCurrent pain and headache reports2010
Chronic headaches and the neurobiology of somatization.
Review in Current pain and headache reports, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
7 citing papers in PubMed.
- Association between adverse childhood experiences and the risk of developing chronic headache in Chinese individuals: a RCSCD-TCM study in China evidence from a national survey.BMC public health · 2024Article
- Engagement with emotional concerns in general practice: a thematic analysis of GP consultations.BJGP open · 2024Article
- [Neurobiology of posttraumatic and somatoform disorders in the elderly].Zeitschrift fur Gerontologie und Geriatrie · 2014Article
- Occupational Stress and Physical Symptoms among Family Medicine Residents.Korean journal of family medicine · 2013Article
- Functional network connectivity of pain-related resting state networks in somatoform pain disorder: an exploratory fMRI study.Journal of psychiatry & neuroscience : JPN · 2013Article
- [Etiopathogenetic aspects of somatoform disorders].Der Nervenarzt · 2012Article
- Consultation-liaison psychiatry for patients with headaches.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2011Article
Corrections and comments
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Authors and funding
1 author.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Pain sensitivity is an adaptive process affected by expectation, mood, coping, operant conditioning, and the preconscious allocation of attention. Underlying mechanisms may include encoding of similar experiences (eg, depression, loss, pain-distress) in overlapping patterns of activation, failure of common regulatory mechanisms, direct top-down activation of the pain matrix, and changes in descending pain facilitatory and inhibitory tone. In theory, the combination of glial cell activation from psychological stress and neural firing from nociceptive input may be particularly likely to lead to pain sensitization and long-term structural changes in pain processing regions of the brain. In these ways, headaches in which chronicity, diffuseness, and distress seem better accounted for by psychological than by medical variables can be understood in neurobiological terms. This can allow psychological treatment of physical distress to be objective, nonthreatening, and relatively precise.
Indexed as
Identifiers
20425215What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.