Evidence map›Paper›PMID 20348961›Full record

ArticleNature genetics2010

Unlocking the pathogenesis of celiac disease.

Robert M Plenge

Abstract readCommentNews
PubMed Publisher
In one paragraph

Article in Nature genetics, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Review
  2. Effect of wheat grain protein composition on end-use quality.Journal of food science and technology · 2020
    Review
  3. Zonulin, regulation of tight junctions, and autoimmune diseases.Annals of the New York Academy of Sciences · 2012
    Review
  4. Leaky gut and autoimmune diseases.Clinical reviews in allergy & immunology · 2012
    Review
  5. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

1 author.

Robert M Plenge

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

A genome-wide association study reports more than a dozen new susceptibility loci for celiac disease. Analysis of eQTL data from these and previously established risk loci sheds light on the genetic pathways underlying this common autoimmune disease.

Indexed as

Genetic Predisposition to DiseasePolymorphism, Single NucleotideCeliac DiseaseGenome-Wide Association StudyHumansRisk Factors

Identifiers

PMID20348961

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.