ArticleJournal of bacteriology2010
SprB is the molecular link between Salmonella pathogenicity island 1 (SPI1) and SPI4.
Article in Journal of bacteriology, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.
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Who cites it
14 citing papers in PubMed.
- Review
- Article
- Two Additional Connections between the Transcriptional Programs Controlling Invasion and Intracellular Replication of Salmonella: HilD-SprB Positively RegulatesJournal of bacteriology · 2022Article
- Prophage-mediated genome differentiation of theMicrobial genomics · 2022Article
- HilD induces expression of a novel Salmonella Typhimurium invasion factor, YobH, through a regulatory cascade involving SprB.Scientific reports · 2019Article
- Inorganic Polyphosphate Is Essential forFrontiers in cellular and infection microbiology · 2018Article
- Article
- Multiple adaptive routes of Salmonella enterica Typhimurium to biocide and antibiotic exposure.BMC genomics · 2016Article
- Mathematical model of flagella gene expression dynamics in Salmonella enterica serovar typhimurium.Systems and synthetic biology · 2015Article
- Identification of HilD-regulated genes in Salmonella enterica serovar Typhimurium.Journal of bacteriology · 2014Article
- Gene expression analysis of Salmonella enterica SPI in macrophages indicates differences between serovars that induce systemic disease from those normally causing enteritis.Veterinary microbiology · 2013Article
- Article
- Role of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genes.Journal of bacteriology · 2010Article
- The role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella.PLoS pathogens · 2010Article
Corrections and comments
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Salmonella pathogenicity island 1 (SPI1) and SPI4 have previously been shown to be jointly regulated. We report that SPI1 and SPI4 gene expression is linked through a transcriptional activator, SprB, encoded within SPI1 and regulated by HilA. SprB directly activates SPI4 gene expression and weakly represses SPI1 gene expression through HilD.
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Registered trials
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