Can scarring be turned off?

Jeffrey M Davidson

Abstract readComment

In one paragraph

Article in The American journal of pathology, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it

0cells of the map it votes in

4citing papers in PubMed

–field-weighted citation impact

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

Skin tissue repair: Matrix microenvironmental influences.Matrix biology : journal of the International Society for Matrix Biology · 2016
Review
Chemokines in Wound Healing and as Potential Therapeutic Targets for Reducing Cutaneous Scarring.Advances in wound care · 2015
Review
Delayed cutaneous wound closure in HO-2 deficient mice despite normal HO-1 expression.Journal of cellular and molecular medicine · 2014
Article
Fibronectin binding modulates CXCL11 activity and facilitates wound healing.PloS one · 2013
Article

4 · The record

Corrections and comments

Comment on
Lack of CXC chemokine receptor 3 signaling leads to hypertrophic and hypercellular scarring.2010

5 · Who and what money

Authors and funding

1 author.

Jeffrey M DavidsonDepartment of Pathology, Vanderbilt University School of Medicine, Nashville, TN 37232-2562, USA. jeff.davidson@vanderbilt.edu

Funding

Skin Regeneration with Stem Cells and ScaffoldsR01AR056138 · NIAMS · VANDERBILT UNIVERSITY · PI DAVIDSON, JEFFREY M., GUELCHER, SCOTT A · 2009 to 2013

$3.3M

ELASTIN AND COLLAGEN IN THE AGING PROCESSR01AG006528 · NIA · VANDERBILT UNIVERSITY · PI DAVIDSON, JEFFREY M. · 1986 to 2011

$2.9M

CARP: Angiogenic Gene Therapy in Diabetic WoundsR01DK065656 · NIDDK · VANDERBILT UNIVERSITY · PI DAVIDSON, JEFFREY M. · 2003 to 2011

$2.3M

NIAMS NIH HHS AR056138NIAMS NIH HHS R01 AR056138NIA NIH HHS AG06528NIA NIH HHS R01 AG006528NIDDK NIH HHS DK065656NIDDK NIH HHS R01 DK065656

6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

CicatrixInflammationWound HealingAnimalsEpidermal Growth FactorFibrosisHumansInterferon-gammaInterleukin-10LigandsMiceModels, BiologicalReceptors, CXCR3Signal TransductionStress, MechanicalCXCR3 protein, humanEpidermal Growth FactorIL10 protein, humanInterferon-gammaInterleukin-10LigandsReceptors, CXCR3

Identifiers

PMID20185579

DOI10.2353/ajpath.2010.100064

PMCPMC2843449

What OpenQuestion holds

Textmetadata

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.