Evidence map›Paper›PMID 20151329›Full record

ArticleNeuroscience and behavioral physiology2010

Dynamics of cognitive anomalies in patients with first episodes of juvenile endogenous psychosis.

V G Kaleda, I S Lebedeva, A N Barkhatova, M A Omel'chenko, S A Golubev, M A Sidorova

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Article in Neuroscience and behavioral physiology, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

V G KaledaScientific Center for Mental Health, Russian Academy of Medical Sciences, Moscow, Russia.
I S Lebedeva
A N Barkhatova
M A Omel'chenko
S A Golubev
M A Sidorova

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The therapeutic dynamics of neuropsychological and neurophysiological markers of impairments to cognitive functions were studied in groups of patients with first episodes of juvenile endogenous psychosis (90 patients). At the initial stage of remission, subjects were found to show improvements in processes associated with voluntary regulation of cognitive functions (due to the activity of extensive networks of cortical and subcortical structures), while more automatic processes (associated mainly with the temporal areas of the brain) remained abnormal. Changes in neurocognitive anomalies during the onset of remission were also identified in groups of patients in whom episodes had different syndromal structures - catatonic, hallucinatory-delusional, and affective-delusional.

Indexed as

AdolescentAdultAffectBrainBrain MappingCatatoniaCognition DisordersHallucinationsHumansMaleNeuropsychological TestsPsychotic DisordersSeverity of Illness IndexVolition

Identifiers

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.