ArticleThe American journal of tropical medicine and hygiene2010
Severe cysticercal meningitis: clinical and imaging characteristics.
Article in The American journal of tropical medicine and hygiene, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
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Who cites it
8 citing papers in PubMed, 31 citations in OpenAlex.
- Taenia solium extracellular vesicles decrease reactive oxygen species production in human neutrophils by inhibiting myeloperoxidase activity.Bioscience reports · 2026Article
- Clinical Symptoms, Imaging Features and Cyst Distribution in the Cerebrospinal Fluid Compartments in Patients with Extraparenchymal Neurocysticercosis.PLoS neglected tropical diseases · 2016Article
- New diagnostic criteria for neurocysticercosis: Reliability and validity.Annals of neurology · 2016Article
- Parasitic diseases of the central nervous system: lessons for clinicians and policy makers.Expert review of neurotherapeutics · 2016Review
- Cranial hypertrophic pachymeningitis secondary to neurocysticercosis.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2013Article
- Extraparenchymal neurocysticercosis in the United States.The American journal of the medical sciences · 2012Review
- Diagnosis and treatment of neurocysticercosis.Nature reviews. Neurology · 2011Review
- Extraparenchymal neurocysticercosis in the United States: a case report.Journal of medical case reports · 2011Article
Corrections and comments
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Authors and funding
5 authors at 3 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
In disease-endemic areas, severe cysticercal meningitis (SCM) is characterized by intense inflammatory cerebrospinal fluid (CSF) and negative bacterial and fungal cultures. There have been no systematic studies of SCM. We characterized patients with SCM and compare them with neurocysticercosis (NC) patients with mild CSF abnormalities by conducting a nine-year retrospective review at a neurological referral center. Two groups of patients were compared: group A, those with severe CSF pleocytosis > 1,000 cells/mm(3) (n = 12), and group B, those with CSF pleocytosis <or= 1,000 cells/mm(3) (n = 126). All patients had positive CSF results in an enzyme-linked immunosorbent assay for cysticercal antigens and negative CSF cultures for bacteria, fungi, and mycobacteria. Intracranial hypertension, meningeal signs, CSF hypoglycorrachia, and a longer clinical course of NC were more frequently seen in group A. It is likely that SCM often goes unrecognized. Its correct identification may reduce morbidity and risks of unnecessary surgery in patients with chronic NC and CSF shunts.
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