Evidence map›Paper›PMID 19875728›Full record

ArticleCirculation research2009

Contributions of ion channel currents to ventricular action potential changes and induction of early afterdepolarizations during acute hypoxia.

Namit Gaur, Yoram Rudy, Livia Hool

Open access · bronzeAbstract read
In one paragraph

Article in Circulation research, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 22 papers.

0numbers the graph read from it
0cells of the map it votes in
22citing papers in PubMed
3.2field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

22 citing papers in PubMed, 49 citations in OpenAlex.

  1. Article
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  4. Sleep-Disordered Breathing Destabilizes Ventricular Repolarization.medRxiv : the preprint server for health sciences · 2024
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  9. Ranolazine is an Effective and Safe Treatment of Adults with Symptomatic Premature Ventricular Contractions due to Triggered Ectopy.The International journal of angiology : official publication of the International College of Angiology, Inc · 2016
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Namit GaurPhysiology M311, School of Biomedical, Biomolecular and Chemical Sciences, The University of Western Australia, 35 Stirling Highway, Crawley, WA 6009, Australia.
Yoram Rudy
Livia Hool
The University of Western Australia · AU

Funding

CARDIAC EXCITATION AND ARRHYTHMIASR01HL049054 · NHLBI · WASHINGTON UNIVERSITY · PI RUDY, YORAM · 1993 to 2017
$5.8M
INVERSE AND FORWARD PROBLEMS IN ELECTROCARDIOGRAPHYR01HL033343 · NHLBI · WASHINGTON UNIVERSITY · PI RUDY, YORAM · 1985 to 2015
$2.9M
INVERSE AND FORWARD PROBLEMS IN ELECTROCARDIOGRAPHYR37HL033343 · NHLBI · WASHINGTON UNIVERSITY · PI RUDY, YORAM · 1998 to 2007
$2.0M
NHLBI NIH HHS R01 HL033343NHLBI NIH HHS R01 HL049054NHLBI NIH HHS R01-HL49054NHLBI NIH HHS R37 HL033343NHLBI NIH HHS R37 HL33343
6 · The paper itself

Abstract

rationaleVariability in delivery of oxygen can lead to electric instability in the myocardium and the generation of arrhythmias. In addition ischemic heart disease and angina are associated with an increase in circulating catecholamines that further increases the risk of developing ventricular tachyarrhythmias.

objectiveWe investigated the net effects of acute hypoxia and catecholamines on the cardiac action potential. METHODS AND

resultsWe incorporated all published data on the effects of hypoxia on the late Na(+) current (I(Na-L)), the fast Na(+) current (I(Na)), the basal L-type Ca(2+) channel current (I(Ca-L)), and the slow (I(Ks)) and rapid components of the delayed rectifier K(+)-current (I(Kr)) in the absence and presence of beta-adrenergic receptor (beta-AR) stimulation into the Luo-Rudy model of the action potential. Hypoxia alone had little effect on the action potential configuration or action potential duration. However in the presence of beta-AR stimulation, hypoxia caused a prolongation of the action potential and early afterdepolarizations (EADs) and spontaneous tachycardia were induced. Experiments performed in guinea pig ventricular myocytes confirmed the modeling results.

conclusionsEADs occur predominantly because of the increased sensitivity of I(Ca-L) to beta-AR stimulation during hypoxia. beta-AR stimulation is necessary to induce EADs as EADs are never observed during hypoxia in the absence of beta-AR stimulation.

Indexed as

Calcium SignalingAction PotentialsAdrenergic beta-AgonistsAnimalsArrhythmias, CardiacCalcium Channels, L-TypeCell HypoxiaDelayed Rectifier Potassium ChannelsDose-Response Relationship, DrugGuinea PigsHeart VentriclesIsoproterenolModels, CardiovascularMyocytes, CardiacOxygenPatch-Clamp TechniquesAdrenergic beta-AgonistsCalcium Channels, L-TypeDelayed Rectifier Potassium ChannelsIsoproterenolOxygenReceptors, Adrenergic, betaSodium Channels

Identifiers

PMID19875728
PMCPMC2826175
OpenAlexW2152074480

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.