Evidence map›Paper›PMID 19865174›Full record

ArticleNature2009

Exploitation of binding energy for catalysis and design.

Summer B Thyme, Jordan Jarjour, Ryo Takeuchi, James J Havranek, Justin Ashworth, Andrew M Scharenberg, Barry L Stoddard, David Baker

Abstract read
In one paragraph

Article in Nature, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 55 papers.

0numbers the graph read from it
0cells of the map it votes in
55citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

55 citing papers in PubMed.

  1. Article
  2. Computational design of sequence-specific DNA-binding proteins.Nature structural & molecular biology · 2025
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Summer B ThymeDepartment of Biochemistry, University of Washington, Seattle, Washington 98195, USA. sthyme@u.washington.edu
Jordan Jarjour
Ryo Takeuchi
James J Havranek
Justin Ashworth
Andrew M Scharenberg
Barry L Stoddard
David Baker

Funding

Directed Protein Evolution for Design of LAGLIDADGs with Novel Recognition SpeciRL1CA133832 · NCI · SEATTLE CHILDREN'S HOSPITAL · PI SCHARENBERG, ANDREW M. · 2007 to 2011
$2.2M
Computational Design of an LAGLIDADGs with Novel Recognition Specificities (CompoRL1GM084433 · NIGMS · UNIVERSITY OF WASHINGTON · PI BAKER, DAVID · 2007 to 2011
$1.9M
Computational Modeling and Design of Specific Protein-DNA InterfacesR00RR024107 · NCRR · WASHINGTON UNIVERSITY · PI HAVRANEK, JAMES J · 2008 to 2010
$742k
Howard Hughes Medical InstituteNCI NIH HHS RL1 CA133832NCI NIH HHS RL1CA133832NCRR NIH HHS R00 RR024107NIGMS NIH HHS GM084433NIGMS NIH HHS RL1 GM084433
6 · The paper itself

Abstract

Enzymes use substrate-binding energy both to promote ground-state association and to stabilize the reaction transition state selectively. The monomeric homing endonuclease I-AniI cleaves with high sequence specificity in the centre of a 20-base-pair (bp) DNA target site, with the amino (N)-terminal domain of the enzyme making extensive binding interactions with the left (-) side of the target site and the similarly structured carboxy (C)-terminal domain interacting with the right (+) side. Here we show that, despite the approximate twofold symmetry of the enzyme-DNA complex, there is almost complete segregation of interactions responsible for substrate binding to the (-) side of the interface and interactions responsible for transition-state stabilization to the (+) side. Although single base-pair substitutions throughout the entire DNA target site reduce catalytic efficiency, mutations in the (-) DNA half-site almost exclusively increase the dissociation constant (K(D)) and the Michaelis constant under single-turnover conditions (K(M)*), and those in the (+) half-site primarily decrease the turnover number (k(cat)*). The reduction of activity produced by mutations on the (-) side, but not mutations on the (+) side, can be suppressed by tethering the substrate to the endonuclease displayed on the surface of yeast. This dramatic asymmetry in the use of enzyme-substrate binding energy for catalysis has direct relevance to the redesign of endonucleases to cleave genomic target sites for gene therapy and other applications. Computationally redesigned enzymes that achieve new specificities on the (-) side do so by modulating K(M)*, whereas redesigns with altered specificities on the (+) side modulate k(cat)*. Our results illustrate how classical enzymology and modern protein design can each inform the other.

Indexed as

BiocatalysisComputer SimulationThermodynamicsBinding SitesComputational BiologyDNAEndonucleasesKineticsModels, MolecularProtein BindingProtein ConformationRNA-Directed DNA PolymeraseSaccharomyces cerevisiaeSubstrate SpecificityDNAEndonucleasesmaturase I-AniIRNA-Directed DNA Polymerase

Identifiers

PMID19865174
PMCPMC2771326

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.