ArticleCell2009
Breaking up just got easier to do.
Article in Cell, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
12 citing papers in PubMed, 23 citations in OpenAlex.
- Multifaceted Fanconi Anemia Signaling.Trends in genetics : TIG · 2018Review
- Linking the Enzymes that Unlink DNA.Molecular cell · 2013Article
- Joint molecule resolution requires the redundant activities of MUS-81 and XPF-1 during Caenorhabditis elegans meiosis.PLoS genetics · 2013Article
- Involvement of SLX4 in interstrand cross-link repair is regulated by the Fanconi anemia pathway.Proceedings of the National Academy of Sciences of the United States of America · 2011Article
- DNA double-strand break repair in Caenorhabditis elegans.Chromosoma · 2011Review
- Mus81 and Yen1 promote reciprocal exchange during mitotic recombination to maintain genome integrity in budding yeast.Molecular cell · 2010Article
- Rmi1 stimulates decatenation of double Holliday junctions during dissolution by Sgs1-Top3.Nature structural & molecular biology · 2010Article
- Review
- Molecular structures of crossover and noncrossover intermediates during gap repair in yeast: implications for recombination.Molecular cell · 2010Article
- GEN1/Yen1 and the SLX4 complex: Solutions to the problem of Holliday junction resolution.Genes & development · 2010Review
- The Fanconi anemia pathway promotes replication-dependent DNA interstrand cross-link repair.Science (New York, N.Y.) · 2009Article
- Article
Corrections and comments
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Authors and funding
2 authors at 2 institutions in 1 country.
Funding
Abstract
The SLX4 protein functions as a platform for catalytic subunits of structure-specific endonucleases. Findings reported in Cell (Fekairi et al., 2009; Svendsen et al., 2009) and in Molecular Cell (Andersen et al., 2009; Muñoz et al., 2009) now identify the human SLX4 and show that in association with the SLX1 endonuclease it directs the symmetric cleavage and resolution of Holliday junctions.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.