Junctin - the quiet achiever.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

15 citing papers in PubMed.

1. Tubular Aggregate Myopathies: Genetic Heterogeneity and Diverse Clinical Features Converging on Calcium Dysregulation.Cells · 2026
   Review
2. Structural and Functional Regulation of RyR2 in Cardiac Calcium Handling and Arrhythmogenesis.Biomedicines · 2026
   Review
3. Architecture and molecular machinery of skeletal myofibers: a systematic review of the structure-function relationships.Frontiers in cell and developmental biology · 2025
   Review
4. Mutations in proteins involved in E-C coupling and SOCE and congenital myopathies.The Journal of general physiology · 2022
   Review
5. Variants in ASPH cause exertional heat illness and are associated with malignant hyperthermia susceptibility.Nature communications · 2022
   Article
6. The Sarcoplasmic Reticulum of Skeletal Muscle Cells: A Labyrinth of Membrane Contact Sites.Biomolecules · 2022
   Review
7. Ablation of Calsequestrin-1, CaFrontiers in physiology · 2022
   Review
8. Calsequestrin, a key protein in striated muscle health and disease.Journal of muscle research and cell motility · 2021
   Review
9. Calsequestrin: a well-known but curious protein in skeletal muscle.Experimental & molecular medicine · 2020
   Review
10. The couplonopathies: A comparative approach to a class of diseases of skeletal and cardiac muscle.The Journal of general physiology · 2015
    Review
11. A new cytoplasmic interaction between junctin and ryanodine receptor Ca2+ release channels.Journal of cell science · 2015
    Article
12. Age-dependent transcriptome and proteome following transection of neonatal spinal cord of Monodelphis domestica (South American grey short-tailed opossum).PloS one · 2014
    Article
13. High-capacity Ca2+ binding of human skeletal calsequestrin.The Journal of biological chemistry · 2012
    Article
14. Dual role of junctin in the regulation of ryanodine receptors and calcium release in cardiac ventricular myocytes.The Journal of physiology · 2011
    Article
15. Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.The Journal of physiology · 2009
    Article

Corrections and comments

• Commented on by
  Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.2009
• Comment on
  Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis.2009
• Comment on
  Triadin: what possible function 20 years later?2009
• Comment on
  Triadin, not essential, but useful.2009

Authors and funding

3 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as