Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

9 citing papers in PubMed.

1. Exercise Protects Skeletal Muscle Fibers from Age-Related Dysfunctional Remodeling of Mitochondrial Network and Sarcotubular System.Cells · 2026
   Review
2. Cryo-EM Brings the Mechanisms of Activation, Inactivation, and Inhibition of Ryanodine Receptors into Focus.Advances in experimental medicine and biology · 2026
   Review
3. Characterization of a novel zebrafish model of SPEG-related centronuclear myopathy.Disease models & mechanisms · 2022
   Article
4. Communications between Mitochondria and Endoplasmic Reticulum in the Regulation of Metabolic Homeostasis.Cells · 2021
   Review
5. Improper Remodeling of Organelles Deputed to CaInternational journal of molecular sciences · 2021
   Review
6. Congenital myopathies: disorders of excitation-contraction coupling and muscle contraction.Nature reviews. Neurology · 2018
   Review
7. The relationship between form and function throughout the history of excitation-contraction coupling.The Journal of general physiology · 2018
   Review
8. Cardiac Arrhythmias as Manifestations of Nanopathies: An Emerging View.Frontiers in physiology · 2018
   Review
9. Role of ryanodine receptor subtypes in initiation and formation of calcium sparks in arterial smooth muscle: comparison with striated muscle.Journal of biomedicine & biotechnology · 2009
   Review

Corrections and comments

• Comment on
  Deconstructing calsequestrin. Complex buffering in the calcium store of skeletal muscle.2009
• Comment on
  Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles.2009
• Comment on
  Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis.2009
• Comment on
  Triadin: what possible function 20 years later?2009
• Comment on
  Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle.2009
• Comment on
  Calsequestrin-1: a new candidate gene for malignant hyperthermia and exertional/environmental heat stroke.2009
• Comment on
  New roles of calsequestrin and triadin in cardiac muscle.2009
• Comment on
  Cardiac calsequestrin: quest inside the SR.2009
• Comment on
  Store overload-induced Ca2+ release as a triggering mechanism for CPVT and MH episodes caused by mutations in RYR and CASQ genes.2009
• Comment on
  Triadin, not essential, but useful.2009
• Comment on
  Junctin - the quiet achiever.2009
• Comment on
  Calcium entry in skeletal muscle.2009

Authors and funding

2 authors.

Funding

Abstract

PubMed holds no abstract for this paper.

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