Genetics meets epigenetics: HDACs and Wnt signaling in myelin development and regeneration.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

21 citing papers in PubMed.

1. M2-associated microglial states in oligodendrocyte regulation and white matter repair in vascular dementia.Journal of translational medicine · 2026
   Review
2. Histone Modification of Osteogenesis Related Genes Triggered by Substrate Topography Promotes Human Mesenchymal Stem Cell Differentiation.ACS applied materials & interfaces · 2023
   Article
3. CREPT Disarms the Inhibitory Activity of HDAC1 on Oncogene Expression to Promote Tumorigenesis.Cancers · 2022
   Article
4. Osteocalcin attenuates oligodendrocyte differentiation and myelination via GPR37 signaling in the mouse brain.Science advances · 2021
   Article
5. Endocrine-Disrupting Chemicals and Vitamin D Deficiency in the Pathogenesis of Uterine Fibroids.Journal of advanced pharmacy research · 2021
   Article
6. mNeuron · 2020
   Article
7. Histone deacetylase 1 and 2 are essential for murine neural crest proliferation, pharyngeal arch development, and craniofacial morphogenesis.Developmental dynamics : an official publication of the American Association of Anatomists · 2017
   Article
8. Unwrapping Neurotrophic Cytokines and Histone Modification.Cellular and molecular neurobiology · 2017
   Review
9. Developmental exposure to endocrine disrupting chemicals alters the epigenome: Identification of reprogrammed targets.Gynecology and obstetrics research : open journal · 2016
   Article
10. Epigenetics of multiple sclerosis: an updated review.Neuromolecular medicine · 2015
    Review
11. p15RS/RPRD1A (p15INK4b-related sequence/regulation of nuclear pre-mRNA domain-containing protein 1A) interacts with HDAC2 in inhibition of the Wnt/β-catenin signaling pathway.The Journal of biological chemistry · 2015
    Article
12. Polydendrocytes in development and myelin repair.Neuroscience bulletin · 2013
    Review
13. Oligodendrocyte dysfunction in the pathogenesis of amyotrophic lateral sclerosis.Brain : a journal of neurology · 2013
    Article
14. The housekeeping gene hypoxanthine guanine phosphoribosyltransferase (HPRT) regulates multiple developmental and metabolic pathways of murine embryonic stem cell neuronal differentiation.PloS one · 2013
    Article
15. Promoting myelin repair and return of function in multiple sclerosis.FEBS letters · 2011
    Review
16. Shaping the oligodendrocyte identity by epigenetic control.Epigenetics · 2010
    Review
17. Tapping into the glial reservoir: cells committed to remaining uncommitted.The Journal of cell biology · 2010
    Review
18. Biological role of dystroglycan in Schwann cell function and its implications in peripheral nervous system diseases.Journal of biomedicine & biotechnology · 2010
    Review
19. Two-tier transcriptional control of oligodendrocyte differentiation.Current opinion in neurobiology · 2009
    Review
20. Histone deacetylase inhibitors: a chemical genetics approach to understanding cellular functions.Biochimica et biophysica acta
    Review

Corrections and comments

• Comment on
  HDAC1 and HDAC2 regulate oligodendrocyte differentiation by disrupting the beta-catenin-TCF interaction.2009

Authors and funding

2 authors.

Funding

Abstract

PubMed holds no abstract for this paper.

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