Evidence map›Paper›PMID 19525390›Full record

ReviewAmerican journal of physiology. Lung cellular and molecular physiology2009

Promotion of cardiovascular disease by exposure to the air pollutant ozone.

Marsha P Cole, Bruce A Freeman

Abstract readCommentEditorialReview
In one paragraph

Review in American journal of physiology. Lung cellular and molecular physiology, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Article
  2. Article
  3. Bidirectional Associations among Nicotine and Tobacco Smoke, NeuroHIV, and Antiretroviral Therapy.Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology · 2020
    Review
  4. Microglial priming through the lung-brain axis: the role of air pollution-induced circulating factors.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2016
    Article
  5. Article
  6. CD36 mediates endothelial dysfunction downstream of circulating factors induced by O3 exposure.Toxicological sciences : an official journal of the Society of Toxicology · 2013
    Article
  7. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors.

Marsha P Cole
Bruce A Freeman

Funding

Redox-Derived Pulmonary Anti-Inflammatory MediatorsR01HL064937 · NHLBI · UNIVERSITY OF PITTSBURGH AT PITTSBURGH · PI FREEMAN, BRUCE ALAN · 2000 to 2015
$5.6M
NITRIC OXIDE REGULATION OF VASCULAR OXIDANT INJURYR01HL058115 · NHLBI · UNIVERSITY OF PITTSBURGH AT PITTSBURGH · PI FREEMAN, BRUCE ALAN · 1998 to 2007
$3.3M
NHLBI NIH HHS R01-HL-58115NHLBI NIH HHS R01-HL-64937
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

Air PollutantsAtmosphereCardiovascular DiseasesHumansOzoneSunlightAir PollutantsOzone

Identifiers

PMID19525390
PMCPMC2742797

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.