Evidence map›Paper›PMID 19365138›Full record

ArticleHuman heredity2009

The effect of inbreeding on the distribution of compound heterozygotes: a lesson from Lipase H mutations in autosomal recessive woolly hair/hypotrichosis.

Lynn Petukhova, Yutaka Shimomura, Muhammad Wajid, Prakash Gorroochurn, Susan E Hodge, Angela M Christiano

Open access · bronzeAbstract read
In one paragraph

Article in Human heredity, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.9field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 35 citations in OpenAlex.

  1. Review
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  3. Splice modulation strategy applied to deep intronic variants inProceedings of the National Academy of Sciences of the United States of America · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 2 countries.

Lynn PetukhovaDepartment of Dermatology, School of Public Health, Columbia University, New York, N.Y. 10032, USA.
Yutaka Shimomura
Muhammad Wajid
Prakash Gorroochurn
Susan E Hodge
Angela M Christiano
Columbia University · USInstitut thématique Génétique, génomique et bioinformatique · FR

Funding

MOLECULAR GENETICS OF THE KERATODERMASR01AR044924 · NIAMS · COLUMBIA UNIVERSITY HEALTH SCIENCES · PI CHRISTIANO, ANGELA M · 1998 to 2008
$2.4M
LINKAGE AND SEGREGATION IN COMPLEX GENETIC DISEASESR01DK031813 · NIDDK · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI HODGE, SUSAN E. · 1986 to 2007
$835k
LINKAGE AND SEGREGATION IN COMPLEX GENETIC DISEASESR55DK031813 · NIDDK · COLUMBIA UNIV NEW YORK MORNINGSIDE · PI HODGE, SUSAN E. · 1991 to 1991
–
NIAMS NIH HHS R01 AR044924NIAMS NIH HHS R01AR44924NIDDK NIH HHS DK-31813NIDDK NIH HHS R01 DK031813
6 · The paper itself

Abstract

Autozygosity mapping in consanguineous families has proven to be a powerful method for identifying recessive disease genes. Using this technique with whole genome SNP data generated from low density mapping arrays, we previously identified two genes that underlie autosomal recessive woolly hair (ARWH/hypotrichosis; OMIM278150), specifically P2RY5 and Lipase H (LIPH). In the current study, we sought to identify a novel disease locus for ARWH/hypotrichosis by analyzing two large consanguineous families from Pakistan who had initially been excluded for mutations at either of these disease loci by haplotype analysis with microsatellite markers. A genome-wide analysis of 10 members from each of the two families failed to identify significant regions of autozygosity or linkage. Upon genotyping an additional 10 family members in one of the families, parametric linkage analysis identified a region on chromosome 3q27 with evidence for linkage (Z = 2.5). Surprisingly, this region contains the LIPH gene. Microsatellite markers located within the LIPH gene were used for haplotype analysis and demonstrated that not one, but two haplotypes were segregating with the phenotype in each of these families. DNA sequencing identified two distinct LIPH mutations (280_369dup90 and 659_660delTA). Each affected individual (n = 38) was either homozygous for one mutation (n = 7 and 16 respectively), or compound heterozygous (n = 15). A review of the literature identified several reports of compound heterozygotes in consanguineous families. Prompted by this finding, we derived the probability that a patient affected with a recessive disease is carrying two mutations at the disease locus. We suggest that the validity of the IBD assumption may be challenged in large consanguineous families.

Indexed as

ConsanguinityGenes, RecessiveHeterozygoteMutationAmino Acid SequenceBase SequenceChromosomes, Human, Pair 3DNAFemaleHaplotypesHumansHypotrichosisLipaseMaleMicrosatellite RepeatsMolecular Sequence DataDNALipaseLIPC protein, human

Identifiers

PMID19365138
PMCPMC2874739
OpenAlexW2057302300

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.