ReviewCurrent atherosclerosis reports2009
Genetics of stroke.
Review in Current atherosclerosis reports, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
4 citing papers in PubMed.
- Article
- Natural genetic variation in Stim1 creates stroke in the spontaneously hypertensive rat.Genes and immunity · 2020Article
- Association of protein Z and factor VII gene polymorphisms with risk of cerebral hemorrhage: a case-control and a family-based association study in a Chinese Han population.Journal of genetics · 2016Article
- SigCS base: an integrated genetic information resource for human cerebral stroke.BMC systems biology · 2011Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
Abstract
Stroke is the third leading cause of death and the leading cause of severe long-term disability in developed countries. Despite significant progress in understanding the risk factors conferring disease predisposition, the genetic and molecular basis of stroke remains poorly understood. Recent advances in the identification and characterization of patterns of DNA sequence variation in human populations hold the promise that stroke genomics will offer significant insights into disease pathophysiology and open new avenues for the development of novel therapeutic modalities. However, beyond single nucleotide polymorphisms, the emergence of additional sources of genomic variability as major factors in disease etiology is likely to transform our DNA-centric approaches toward more integrative and comprehensive strategies. This review provides an overview of the current progress and future prospects of the application of genomic sciences to stroke research.
Indexed as
Identifiers
19361347What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.