ReviewCurrent psychiatry reports2009
Advances in genetic studies of attention-deficit/hyperactivity disorder.
Review in Current psychiatry reports, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
17 citing papers in PubMed.
- Trial
- Inhibition of stress resilience and adult hippocampal neurogenesis by platelet-derived LPA16:0 in anxiety.Nature communications · 2026Article
- Developmental pyrethroid exposure disrupts molecular pathways for MAP kinase and circadian rhythms in mouse brain.Physiological genomics · 2025Article
- Attention-deficit/hyperactivity disorder (ADHD) in adults: a multilayered approach to a serious disorder of inattention to the future.Arquivos de neuro-psiquiatria · 2024Review
- Developmental pyrethroid exposure disrupts molecular pathways for MAP kinase and circadian rhythms in mouse brain.bioRxiv : the preprint server for biology · 2024Article
- Identification of factors associated with the efficacy of atomoxetine in adult attention-deficit/hyperactivity disorder.Neuropsychopharmacology reports · 2022Article
- Review
- Epigenetic Programming by Maternal Behavior in the Human Infant.Pediatrics · 2018Article
- DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?Environmental epigenetics · 2016Article
- Multifactorial Origin of Neurodevelopmental Disorders: Approaches to Understanding Complex Etiologies.Toxics · 2015Review
- Lead, mercury, and cadmium exposure and attention deficit hyperactivity disorder in children.Environmental research · 2013Article
- A genetic association study of DNA methylation levels in the DRD4 gene region finds associations with nearby SNPs.Behavioral and brain functions : BBF · 2012Article
- Attention deficit/hyperactivity disorder-derived coding variation in the dopamine transporter disrupts microdomain targeting and trafficking regulation.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2012Article
- Assessment of physician practices in adult attention-deficit/hyperactivity disorder.The primary care companion for CNS disorders · 2012Article
- Attention deficit/hyperactivity disorder: a focused overview for children's environmental health researchers.Environmental health perspectives · 2010Review
- Lead and PCBs as risk factors for attention deficit/hyperactivity disorder.Environmental health perspectives · 2010Review
- Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD.Attention deficit and hyperactivity disorders · 2010Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Attention-deficit/hyperactivity disorder (ADHD) is among the most common childhood-onset psychiatric disorders. Although family, twin, and adoption studies demonstrate that ADHD is a highly heritable condition, studies also suggest that genetic architecture is complex, prompting the use of more advanced methodologies such as genome-wide linkage and association studies. Although such studies are theoretically compelling, replication of these results has been inconsistent. Meta-analyses have produced more reliable results, but the associations identified to date account for only a small percentage of the genetic component of ADHD. Approaches such as neuroimaging genetics and epigenetic studies are being explored to probe further the etiologic complexity of this disorder.
Indexed as
Identifiers
19302768What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.