When integrins fail to integrate.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

4 citing papers in PubMed.

1. The thymus gland is a target and maintainer of amyotrophic lateral sclerosis pathophysiology.Brain, behavior, & immunity - health · 2026
   Article
2. Integrin VLA-4 as a PET imaging biomarker of hyper-adhesion in transgenic sickle mice.Blood advances · 2020
   Article
3. Lessons from rare maladies: leukocyte adhesion deficiency syndromes.Current opinion in hematology · 2013
   Review
4. Signal transduction during activation and inhibition of natural killer cells.Current protocols in immunology · 2010
   Review

Corrections and comments

• Comment on
  A point mutation in KINDLIN3 ablates activation of three integrin subfamilies in humans.2009
• Comment on
  Kindlin-3 is required for beta2 integrin-mediated leukocyte adhesion to endothelial cells.2009
• Comment on
  Leukocyte adhesion deficiency-III is caused by mutations in KINDLIN3 affecting integrin activation.2009

Authors and funding

2 authors.

Funding

Abstract

PubMed holds no abstract for this paper.

Indexed as