ArticleThe Journal of cell biology1991
The structural basis for the intrinsic disorder of the actin filament: the "lateral slipping" model.
Article in The Journal of cell biology, 1991. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 32 papers.
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Who cites it
32 citing papers in PubMed, 118 citations in OpenAlex.
- Reaching the full potential of cryo-EM reconstructions with molecular dynamics simulations at 310 K: Actin filaments as an example.Proceedings of the National Academy of Sciences of the United States of America · 2025Article
- From stress fiber to focal adhesion: a role of actin crosslinkers in force transmission.Frontiers in cell and developmental biology · 2024Review
- Mechanism of actin filament nucleation.Biophysical journal · 2021Article
- Insights into Actin Polymerization and Nucleation Using a Coarse-Grained Model.Biophysical journal · 2020Article
- Glucose starvation triggers filamentous septin assemblies in anBiology open · 2019Article
- Helical rotation of the diaphanous-related formin mDia1 generates actin filaments resistant to cofilin.Proceedings of the National Academy of Sciences of the United States of America · 2018Article
- Two deafness-causing (DFNA20/26) actin mutations affect Arp2/3-dependent actin regulation.The Journal of biological chemistry · 2012Article
- Intermittent depolymerization of actin filaments is caused by photo-induced dimerization of actin protomers.Proceedings of the National Academy of Sciences of the United States of America · 2012Article
- A novel approach to the structural analysis of partially decorated actin based filaments.Journal of structural biology · 2010Article
- Methods for identifying and averaging variable molecular conformations in tomograms of actively contracting insect flight muscle.Journal of structural biology · 2009Article
- Actin disassembly by cofilin, coronin, and Aip1 occurs in bursts and is inhibited by barbed-end cappers.The Journal of cell biology · 2008Article
- Glycolytic enzyme interactions with yeast and skeletal muscle F-actin.Biophysical journal · 2006Article
- Actin-destabilizing factors disrupt filaments by means of a time reversal of polymerization.Proceedings of the National Academy of Sciences of the United States of America · 2004Article
- Mammalian cardiac muscle thick filaments: their periodicity and interactions with actin.Biophysical journal · 2002Article
- Ca(2+)-induced movement of tropomyosin in skeletal muscle thin filaments observed by multi-site FRET.Biophysical journal · 2002Article
- Sound attenuation of polymerizing actin reflects supramolecular structures: viscoelastic properties of actin gels modified by cytochalasin D, profilin and alpha-actinin.The Biochemical journal · 2001Article
- Distinct structural changes detected by X-ray fiber diffraction in stabilization of F-actin by lowering pH and increasing ionic strength.Biophysical journal · 2001Article
- Actin and the smooth muscle regulatory proteins: a structural perspective.Journal of muscle research and cell motility · 2000Review
- Cryoatomic force microscopy of filamentous actin.Biophysical journal · 2000Article
- Evidence for a conformational change in actin induced by fimbrin (N375) binding.The Journal of cell biology · 1997Article
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6 authors at 1 institution in 1 country.
Funding
Abstract
Three-dimensional (3-D) helical reconstructions computed from electron micrographs of negatively stained dispersed F-actin filaments invariably revealed two uninterrupted columns of mass forming the "backbone" of the double-helical filament. The contact between neighboring subunits along the thus defined two long-pitch helical strands was spatially conserved and of high mass density, while the intersubunit contact between them was of lower mass density and varied among reconstructions. In contrast, phalloidinstabilized F-actin filaments displayed higher and spatially more conserved mass density between the two long-pitch helical strands, suggesting that this bicyclic hepta-peptide toxin strengthens the intersubunit contact between the two strands. Consistent with this distinct intersubunit bonding pattern, the two long-pitch helical strands of unstabilized filaments were sometimes observed separated from each other over a distance of two to six subunits, suggesting that the intrastrand intersubunit contact is also physically stronger than the interstrand contact. The resolution of the filament reconstructions, extending to 2.5 nm axially and radially, enabled us to reproducibly "cut out" the F-actin subunit which measured 5.5 nm axially by 6.0 nm tangentially by 3.2 nm radially. The subunit is distinctly polar with a massive "base" pointing towards the "barbed" end of the filament, and a slender "tip" defining its "pointed" end (i.e., relative to the "arrowhead" pattern revealed after stoichiometric decoration of the filaments with myosin subfragment 1). Concavities running approximately parallel to the filament axis both on the inner and outer face of the subunit define a distinct cleft separating the subunit into two domains of similar size: an inner domain confined to radii less than or equal to 2.5-nm forms the uninterrupted backbone of the two long-pitch helical strands, and an outer domain placed at radii of 2-5-nm protrudes radially and thus predominantly contributes to the outer part of the massive base. Quantitative evaluation of successive crossover spacings along individual F-actin filaments revealed the deviations from the mean repeat to be compensatory, i.e., short crossovers frequently followed long ones and vice versa. The variable crossover spacings and diameter of the F-actin filament together with the local unraveling of the two long-pitch helical strands are explained in terms of varying amounts of compensatory "lateral slipping" of the two strands past each other roughly perpendicular to the filament axis. This intrinsic disorder of the actin filament may enable the actin moiety to play a more active role in actin-myosin-based force generation than merely act as a rigid passive cable as has hitherto been assumed.
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