ReviewCurrent neurology and neuroscience reports2009
Inclusion body myositis: review of recent literature.
Review in Current neurology and neuroscience reports, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
11 citing papers in PubMed.
- Amyloid deposits and inflammatory infiltrates in sporadic inclusion body myositis: the inflammatory egg comes before the degenerative chicken.Acta neuropathologica · 2015Review
- Formation of gelsolin amyloid fibrils in the rough endoplasmic reticulum of skeletal muscle in the gelsolin mouse model of inclusion body myositis: comparative analysis to human sporadic inclusion body myositis.Ultrastructural pathology · 2013Article
- A Caenorhabditis elegans model system for amylopathy study.Journal of visualized experiments : JoVE · 2013Article
- From soluble aβ to progressive aβ aggregation: could prion-like templated misfolding play a role?Brain pathology (Zurich, Switzerland) · 2013Review
- Inclusion body myositis: therapeutic approaches.Degenerative neurological and neuromuscular disease · 2012Review
- Expression of human amyloid precursor protein in the skeletal muscles of Drosophila results in age- and activity-dependent muscle weakness.BMC physiology · 2011Article
- Inflammatory myopathies.Current treatment options in neurology · 2011Article
- Sporadic inclusion body myositis: possible pathogenesis inferred from biomarkers.Current opinion in neurology · 2010Review
- Theories of the pathogenesis of inclusion body myositis.Current rheumatology reports · 2010Review
- Clinical applications of intravenous immunoglobulins in neurology.Clinical and experimental immunology · 2009Review
- Vacuolar myopathy in a dog resembling human sporadic inclusion body myositis.Acta neuropathologica · 2009Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
Abstract
Inclusion body myositis (IBM) is a progressive inflammatory skeletal muscle disease of unknown cause and without effective treatment. This article discusses existing literature, emphasizing disease mechanisms and models. In particular, it addresses limitations in the beta-amyloid-mediated theory of IBM myofiber injury, flawed rationales of animal models of this disease, and recent reports regarding treatment.
Indexed as
Identifiers
19080758What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.