ReviewNeurotoxicity research2008
Reconsolidation and the fate of consolidated memories.
Review in Neurotoxicity research, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed, 21 citations in OpenAlex.
- Relevance of ERK1/2 Post-retrieval Participation on Memory Processes: Insights in Their Particular Role on Reconsolidation and Persistence of Memories.Frontiers in molecular neuroscience · 2019Review
- Baclofen facilitates the extinction of methamphetamine-induced conditioned place preference in rats.Behavioral neuroscience · 2011Article
- Cognitive symptoms facilitatory for diagnoses in neuropsychiatric disorders: executive functions and locus of control.Neurotoxicity research · 2008Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The predominant view about memory formation states that a consolidation process stabilizes newly acquired traces until they are safely stored in the brain. However, during the last ten years evidence has accumulated to indicate that, upon retrieval, consolidated memories are rendered again vulnerable to the action of metabolic blockers, notably protein synthesis inhibitors. This has led to the hypothesis that memories are reconsolidated at the time of retrieval, and that this requires protein synthesis in different brain regions. Here we will address the consolidation-reconsolidation debate and discuss some controversial issues about the reconsolidation hypothesis, in particular the biological role of this process.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.