ReviewJournal of bone and mineral metabolism2009
FGFR3-related dwarfism and cell signaling.
Review in Journal of bone and mineral metabolism, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
18 citing papers in PubMed.
- A Comprehensive Study of De Novo Mutations on the Protein-Protein Interaction Interfaces Provides New Insights into Developmental Delay.Biomolecules · 2022Article
- Precise base editing for theeLife · 2021Article
- Novel and Recurrent Mutations in theMolecular syndromology · 2018Article
- Molecular therapeutic strategies for FGFR3 gene-related skeletal dysplasia.Journal of molecular medicine (Berlin, Germany) · 2017Review
- Pediatric Craniovertebral Junction Surgery.Neurologia medico-chirurgica · 2017Review
- A New Method to Study Heterodimerization of Membrane Proteins and Its Application to Fibroblast Growth Factor Receptors.The Journal of biological chemistry · 2017Article
- Characterization of membrane protein interactions in plasma membrane derived vesicles with quantitative imaging Förster resonance energy transfer.Accounts of chemical research · 2015Article
- Effect of thanatophoric dysplasia type I mutations on FGFR3 dimerization.Biophysical journal · 2015Article
- New evidence for positive selection helps explain the paternal age effect observed in achondroplasia.Human molecular genetics · 2013Article
- Multiple consequences of a single amino acid pathogenic RTK mutation: the A391E mutation in FGFR3.PloS one · 2013Article
- Production of plasma membrane vesicles with chloride salts and their utility as a cell membrane mimetic for biophysical characterization of membrane protein interactions.Analytical chemistry · 2012Article
- Physical-chemical principles underlying RTK activation, and their implications for human disease.Biochimica et biophysica acta · 2012Review
- Direct assessment of the effect of the Gly380Arg achondroplasia mutation on FGFR3 dimerization using quantitative imaging FRET.PloS one · 2012Article
- Article
- Sixteen years and counting: the current understanding of fibroblast growth factor receptor 3 (FGFR3) signaling in skeletal dysplasias.Human mutation · 2012Review
- The A391E mutation enhances FGFR3 activation in the absence of ligand.Biochimica et biophysica acta · 2011Article
- FGFR3 heterodimerization in achondroplasia, the most common form of human dwarfism.The Journal of biological chemistry · 2011Article
- Physical basis behind achondroplasia, the most common form of human dwarfism.The Journal of biological chemistry · 2010Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
PubMed holds no abstract for this paper.
Indexed as
Identifiers
19066716What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.