Evidence map›Paper›PMID 18957543›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2008

Distinct populations of tumor-initiating cells derived from a tumor generated by rat mammary cancer stem cells.

I Zucchi, S Astigiano, G Bertalot, S Sanzone, C Cocola, P Pelucchi, G Bertoli, M Stehling, O Barbieri, A Albertini and 4 more

Open access · greenAbstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.9field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 35 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 5 institutions in 3 countries.

I ZucchiIstituto Tecnologie Biomediche-Consiglio Nazionale delle Richerche, Via Cervi 93, 20090 Segrate-Milan, Italy. ileana.zucchi@itb.cnr.it
S Astigiano
G Bertalot
S Sanzone
C Cocola
P Pelucchi
G Bertoli
M Stehling
O Barbieri
A Albertini
H R Schöler
B G Neel
R A Reinbold
R Dulbecco
Institute of Biomedical Technologies · ITIstituto Scientifico Romagnolo per lo Studio e la Cura dei Tumori · ITBeth Israel Deaconess Medical Center · USEuropean Institute of Oncology · ITSalk Institute for Biological Studies · US

Funding

Telethon GGP04247
6 · The paper itself

Abstract

Tumors derived from rat LA7 cancer stem cells (CSCs) contain a hierarchy of cells with different capacities to generate self-renewing spheres and tubules serially ex vivo and to evoke tumors in vivo. We isolated two morphologically distinct cell types with distinct tumorigenic potential from LA7-evoked tumors: cells with polygonal morphology that are characterized by expression of p21/(WAF1) and p63 and display hallmarks of CSCs and elongated epithelial cells, which generate tumors with far less heterogeneity than LA7 CSCs. Serial transplantation of elongated epithelial cells results in progressive loss of tumorigenic potential; tumor heterogeneity; CD44, E-cadherin, and epithelial cytokeratin expression and increased alpha-smooth muscle actin I and vimentin expression. In contrast, serial transplantation of LA7 CSCs can be performed indefinitely and results in tumors that maintain their heterogeneity, consistent with self-renewal and multilineage differentiation potential. Collectively, our data show that polygonal cells are CSCs, whereas epithelial elongated cells are lineage-committed progenitors with tumorigenic potential, and suggest that tumor progenitors, although lacking indefinite self-renewal potential, nevertheless may make a substantial contribution to tumor development. Because LA7 cells can switch between conditions that favor maintenance of pure CSCs vs. differentiation into other tumor cell types, this cell system provides the opportunity to study factors that influence CSC self-renewal and differentiation. One factor, p63, was identified as a key gene regulating the transition between CSCs and early progenitor cells.

Indexed as

AnimalsCell DifferentiationCell LineageCell Line, TumorClone CellsDisease Models, AnimalFemaleImmunohistochemistryMammary Glands, AnimalMammary Neoplasms, ExperimentalMiceMice, SCIDNeoplasmsNeoplastic Stem CellsRatsStem Cells

Identifiers

PMID18957543
PMCPMC2575224
OpenAlexW1997626619

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.