ArticleJournal of cardiothoracic surgery2008
Protective efficiacy of taurine against pulmonary edema progression: experimental study.
Article in Journal of cardiothoracic surgery, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
7 citing papers in PubMed.
- Hyperoxia arrests pulmonary development in newborn rats via disruption of endothelial tight junctions and downregulation of Cx40.Molecular medicine reports · 2014Article
- Taurine attenuates lung ischemia-reperfusion injury after lung transplantation in rats.Journal of anesthesia · 2014Article
- The efficiency of proanthocyanidin in an experimental pulmonary fibrosis model: comparison with taurine.Inflammation · 2012Article
- Is there any cardioprotective role of Taurine during cold ischemic period following global myocardial ischemia?Journal of cardiothoracic surgery · 2011Article
- The role of oxidative stress and effect of alpha-lipoic acid in reexpansion pulmonary edema - an experimental study.Archives of medical science : AMS · 2010Article
- Reactive species and pulmonary edema.Expert review of respiratory medicine · 2009Article
- Proanthocyanidin to prevent formation of the reexpansion pulmonary edema.Journal of cardiothoracic surgery · 2009Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Re-expansion pulmonary edema (RPE) is an acute, rare and potentially lethal complication. Its beginning is sudden and dramatic. The mechanism is not yet fully understood. Some authors suggest that it may occur after rapid re-inflation of a collapsed lung. It was reported by other authors that it may relate to surfactant depletion or may result from hypoxic capillary damage, leading to increased capillary permeability. In RPE, unilateral lung injury is initiated by cytotoxic oxygen metabolites and temporally associated with an influx of polymorphonuclear neutrophils. These toxic oxygen products are the results of re-oxygenation of a collapsed lung. Treatment of re-expansion pulmonary edema is basically preventive.
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Registered trials
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