ArticleProtein expression and purification2008
High-yield expression in E. coli and refolding of the bZIP domain of activating transcription factor 5.
Article in Protein expression and purification, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
8 citing papers in PubMed, 9 citations in OpenAlex.
- Review
- Self-assembly of the bZIP transcription factor ΔFosB.Current research in structural biology · 2020Article
- Regression/eradication of gliomas in mice by a systemically-deliverable ATF5 dominant-negative peptide.Oncotarget · 2016Article
- claMP Tag: a versatile inline metal-binding platform based on the metal abstraction peptide.Bioconjugate chemistry · 2014Article
- Human cytochrome P450 17A1 conformational selection: modulation by ligand and cytochrome b5.The Journal of biological chemistry · 2014Article
- Influence of the valine zipper region on the structure and aggregation of the basic leucine zipper (bZIP) domain of activating transcription factor 5 (ATF5).Molecular pharmaceutics · 2012Article
- Regulated ATF5 loss-of-function in adult mice blocks formation and causes regression/eradication of gliomas.Oncogene · 2012Article
- Effects of disulfide bond formation and protein helicity on the aggregation of activating transcription factor 5.Molecular pharmaceuticsArticle
Corrections and comments
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
Abstract
Activating transcription factor 5 (ATF5) recently has been demonstrated to play a critical role in promoting the survival of human glioblastoma cells. Interference with the function of ATF5 in an in vivo rat model caused glioma cell death in primary tumors but did not affect the status of normal cells surrounding the tumor, suggesting ATF5 may prove an ideal target for anti-cancer therapy. In order to examine ATF5 as a pharmaceutical target, the protein must be produced and purified to sufficient quantity to begin analyses. Here, a procedure for expressing and refolding the bZIP domain of ATF5 in sufficient yield and final concentration to permit assay development and structural characterization of this target using solution NMR is reported. Two-dimensional NMR and circular dichroism analyses indicate the protein exists in the partially alpha-helical, monomeric x-form conformation with only a small fraction of ATF5 participating in formation of higher-order structure, presumably coiled-coil homodimerization. Despite the persistence of monomers in solution even at high concentration, an electrophoretic mobility shift assay showed that ATF5 is able to bind to the cAMP response element (CRE) DNA motif. Polyacrylamide gel electrophoresis and mass spectrometry were used to confirm that ATF5 can participate in homodimer formation and that this dimerization is mediated by disulfide bond formation.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.