Evidence map›Paper›PMID 18616392›Full record

ReviewAnnual review of psychology2009

Neuropsychological assessment of dementia.

David P Salmon, Mark W Bondi

Open access · greenAbstract readReview
In one paragraph

Review in Annual review of psychology, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 172 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
172citing papers in PubMed, 3 pooled it
7.8field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

172 citing papers in PubMed, 3 syntheses or guidelines pooled it, 425 citations in OpenAlex.

  1. Pooled it
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  6. But What Behavior?ArXiv · 2026
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  19. Recognition Subtests for Form B of the Repeatable Battery for the Assessment of Neuropsychological Status: Preliminary Validation.Archives of clinical neuropsychology : the official journal of the National Academy of Neuropsychologists · 2025
    Observational
  20. Article

112 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

David P SalmonDepartment of Neurosciences, University of California, San Diego, California 92093, USA. dsalmon@ucsd.edu
Mark W Bondi
University of California, San Diego · US

Funding

VIRUS-INDUCED BRAIN PATHOLOGYP50AG005131 · NIA · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI BREWER, JAMES B · 1985 to 2018
$55.4M
Cognitive Abilities of At-Risk Elderly for DementiaR01AG012674 · NIA · VETERANS MEDICAL RESEARCH FDN/SAN DIEGO · PI BONDI, MARK W · 2001 to 2012
$2.7M
COGNITIVE STUDIES OF THE LEWY BODY VARIANT OF ADR01AG012963 · NIA · UNIVERSITY OF CALIFORNIA SAN DIEGO · PI SALMON, DAVID P · 1994 to 2005
$1.0M
Cognitive Abilities of At-Risk Elderly for DementiaR56AG012674 · NIA · VETERANS MEDICAL RESEARCH FDN/SAN DIEGO · PI BONDI, MARK W · 2007 to 2007
$347k
COGNITIVE ABILITIES OF AT RISK ELDERLY FOR DEMENTIAR29AG012674 · NIA · UNIVERSITY OF CALIFORNIA SAN DIEGO · PI BONDI, MARK W · 1994 to 1998
–
NIA NIH HHS AG-05131NIA NIH HHS AG-12674NIA NIH HHS AG-12963NIA NIH HHS P50 AG005131NIA NIH HHS R01 AG012674NIA NIH HHS R01 AG012963NIA NIH HHS R29 AG012674NIA NIH HHS R56 AG012674
6 · The paper itself

Abstract

Neuropsychological studies show that cognitive deficits associated with Alzheimer's disease (AD) are distinct from age-associated cognitive decline. Quantitative and qualitative differences are apparent across many cognitive domains, but are especially obvious in episodic memory (particularly delayed recall), semantic knowledge, and some aspects of executive functions. The qualitatively distinct pattern of deficits is less salient in very old AD patients than in younger AD patients. Although decline in episodic memory is usually the earliest cognitive change that occurs prior to the development of the AD dementia syndrome, asymmetry in cognitive abilities may also occur in this "preclinical" phase of the disease and predict imminent dementia. Discrete patterns of cognitive deficits occur in AD and several neuropathologically distinct age-associated neurodegenerative disorders. Knowledge of these differences helps to clinically distinguish among various causes of dementia and provides useful models for understanding brain-behavior relationships that mediate cognitive abilities affected in various neurodegenerative diseases.

Indexed as

Neuropsychological TestsAgedAlzheimer DiseaseCerebral CortexCognition DisordersDementiaDiagnosis, DifferentialHumansMental RecallNerve NetNeurodegenerative DiseasesRetention, Psychology

Identifiers

PMID18616392
PMCPMC2864104
OpenAlexW2106934216

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.