Evidence map›Paper›PMID 18577513›Full record

ArticleThe Journal of biological chemistry2008

The ubiquitin-protein ligase Nedd4-2 differentially interacts with and regulates members of the Tweety family of chloride ion channels.

Yaowu He, Deanne H Hryciw, Melanie L Carroll, Stephen A Myers, Astrid K Whitbread, Sharad Kumar, Philip Poronnik, John D Hooper

Open access · hybridAbstract read
In one paragraph

Article in The Journal of biological chemistry, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
1.1field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 34 citations in OpenAlex.

  1. Article
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  6. TheFrontiers in molecular neuroscience · 2021
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  15. Role of the ubiquitin system in regulating ion transport.Pflugers Archiv : European journal of physiology · 2011
    Article
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  17. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Yaowu HeInstitute of Health and Biomedical Innovation and School of Life Sciences, Queensland University of Technology, Musk Avenue and Blarney Street, Kelvin Grove, Queensland 4059, Australia.
Deanne H Hryciw
Melanie L Carroll
Stephen A Myers
Astrid K Whitbread
Sharad Kumar
Philip Poronnik
John D Hooper
Queensland University of Technology · AUUniversity of Queensland · AUSouth Australia Pathology · AU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The Tweety proteins comprise a family of chloride ion channels with three members identified in humans (TTYH1-3) and orthologues in fly and murine species. In humans, increased TTYH2 expression is associated with cancer progression, whereas fly Tweety is associated with developmental processes. Structurally, Tweety proteins are characterized by five membrane-spanning domains and N-glycan modifications important for trafficking to the plasma membrane, where these proteins are oriented with the amino terminus located extracellularly and the carboxyl terminus cytoplasmically. In addition to N-glycosylation, ubiquitination mediated by the HECT type E3 ubiquitin ligase Nedd4-2 is a post-translation modification important in regulating membrane proteins. In the present study, we performed a comprehensive analysis of the ability of each of TTYH1-3 to interact with Nedd4-2 and to be ubiquitinated and regulated by this ligase. Our data indicate that Nedd4-2 binds to two family members, TTYH2 and TTYH3, which contain consensus PY ((L/P)PXY) binding sites for HECT type E3 ubiquitin ligases, but not to TTYH1, which lacks this motif. Consistently, Nedd4-2 ubiquitinates both TTYH2 and TTYH3. Importantly, we have shown that endogenous TTYH2 and Nedd4-2 are binding partners and demonstrated that the TTYH2 PY motif is essential for these interactions. We have also shown that Nedd4-2-mediated ubiquitination of TTYH2 is a critical regulator of cell surface and total cellular levels of this protein. These data, indicating that Nedd4-2 differentially interacts with and regulates TTYH1-3, will be important for understanding mechanisms controlling Tweety proteins in physiology and disease.

Indexed as

Amino Acid MotifsAnimalsBinding SitesCell MembraneChloride ChannelsCHO CellsCricetinaeCricetulusEndosomal Sorting Complexes Required for TransportGlycosylationHumansMembrane ProteinsNedd4 Ubiquitin Protein LigasesNeoplasm ProteinsProtein Processing, Post-TranslationalUbiquitinChloride ChannelsEndosomal Sorting Complexes Required for TransportMembrane ProteinsNedd4L protein, humanNedd4 protein, humanNedd4 Ubiquitin Protein LigasesNeoplasm ProteinsTTYH1 protein, humanTTYH2 protein, humanTTYH3 protein, humanUbiquitinUbiquitin-Protein Ligases

Identifiers

PMID18577513
PMCPMC3259790
OpenAlexW2079028932

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.