ArticleMaternal and child health journal2009
Fetal exposure to secondhand tobacco smoke assessed by maternal self-reports and cord blood cotinine: prospective cohort study in Krakow.
Article in Maternal and child health journal, 2009. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.
What it found
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Who cites it
12 citing papers in PubMed, 21 citations in OpenAlex.
- Prenatal and Childhood Smoke Exposure Associations with Cognition, Language, and Attention-Deficit/Hyperactivity Disorder.The Journal of pediatrics · 2023Article
- Tobacco Retail Outlets, Neighborhood Deprivation and the Risk of Prenatal Smoke Exposure.Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco · 2022Article
- Neighborhood Deprivation is Associated with Increased Risk of Prenatal Smoke Exposure.Prevention science : the official journal of the Society for Prevention Research · 2022Article
- Nicotine and Its Downstream Metabolites in Maternal and Cord Sera: Biomarkers of Prenatal Smoking Exposure Associated with Offspring DNA Methylation.International journal of environmental research and public health · 2020Article
- Non-smoking pregnant women and their fetuses are exposed to environmental tobacco smoke as a result of living in multiunit housing.Journal of exposure science & environmental epidemiology · 2017Article
- Second hand smoke exposure in public venues and mental disorder: a representative nationwide study of China.Tobacco induced diseases · 2015Article
- Assessing secondhand smoke exposure with reported measures.Tobacco control · 2013Review
- Assessment of prenatal exposure to tobacco smoke by cotinine in cord blood for the evaluation of smoking control policies in Spain.BMC pregnancy and childbirth · 2012Article
- Exposure assessment in cohort studies of childhood asthma.Environmental health perspectives · 2011Article
- A prospective cohort study of biomarkers of prenatal tobacco smoke exposure: the correlation between serum and meconium and their association with infant birth weight.Environmental health : a global access science source · 2010Article
- Prenatal exposure to PBDEs and neurodevelopment.Environmental health perspectives · 2010Article
- The effects of prenatal secondhand smoke exposure on preterm birth and neonatal outcomes.Journal of obstetric, gynecologic, and neonatal nursing : JOGNNArticle
Corrections and comments
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Authors and funding
9 authors at 3 institutions in 2 countries.
Funding
Abstract
objectivesWhile the validity of self-reported smoking habits is generally judged as satisfactory, objective markers of secondhand smoke (SHS) exposure may be more useful in validating the causal links between prenatal SHS and health effects. The cohort study in Krakow provided an opportunity for comparative assessment of fetal exposure to SHS based upon questionnaires and cord blood cotinine measurements.
methodsThe study sample included 467 newborns born to women recruited in the first and second trimester of pregnancy. To compare the validity of self-reported SHS and cord blood cotinine levels in assessing the association between fetal passive smoking and health effects of newborns, we separately examined the regression coefficients of birthweight on self-reported number of cigarettes smoked by other household members during the entire pregnancy and cord blood cotinine levels.
resultsIn the non-exposed newborns the geometric mean of cord blood cotinine was 0.077 ng/ml and was significantly lower than in newborns with a maternal report of SHS. Cord cotinine levels were more highly correlated with a self-reported number of cigarettes smoked daily at home in the third trimester of pregnancy. The two measures of SHS (number of cigarettes and number of hours of daily exposure) were equally well correlated with cord blood cotinine levels. Using cotinine as the exposure variable, overall the association was not significant; but among the subgroup with cord cotinine levels above the median (> or =0.083 ng/ml), the association with birthweight was significant (beta coefficient = -113.65, P = 0.041).
conclusionThe study provides evidence that the assessment of fetal SHS exposure based on cord blood cotinine produced better estimates of the association between exposure and birth outcomes.
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