ReviewSeminars in nephrology2008
Bone disease and idiopathic hypercalciuria.
Review in Seminars in nephrology, 2008. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
16 citing papers in PubMed.
- Endocrine-related osteoporosis: the state of the art.Frontiers in endocrinology · 2026Review
- Vitamin D and Calcium Supplementation and Urolithiasis: A Controversial and Multifaceted Relationship.Nutrients · 2023Review
- Morbidity Associated with Chronic Hyponatremia.Journal of clinical medicine · 2023Article
- The Urine Calcium/Creatinine Ratio and Uricemia during Hyponatremia of Different Origins: Clinical Implications.Journal of clinical medicine · 2023Article
- Idiopathic osteoporosis, Ehlers-Danlos syndrome, postural orthostatic tachycardia syndrome, and mast cell activation disorder in a 27-year-old male patient: A unique case presentation.Clinical case reports · 2022Article
- LIFESTYLE RISK FACTORS AND BONE MASS IN RECURRENT STONE-FORMING PATIENTS: A CROSS-SECTIONAL STUDY IN 144 SUBJECTS.Acta clinica Croatica · 2019Article
- Skeletal Consequences of Nephropathic Cystinosis.Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research · 2018Article
- Syndrome of inappropriate anti-diuresis induces volume-dependent hypercalciuria.Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA · 2017Observational
- Pediatric primary urolithiasis: Symptoms, medical management and prevention strategies.World journal of nephrology · 2015Article
- Calcium renal lithiasis: metabolic diagnosis and medical treatment.Sao Paulo medical journal = Revista paulista de medicina · 2013Review
- Idiopathic hypercalciuria and bone health.Current osteoporosis reports · 2012Review
- Plasma and urinary levels of cytokines in patients with idiopathic hypercalciuria.Pediatric nephrology (Berlin, Germany) · 2012Article
- The epithelial sodium/proton exchanger, NHE3, is necessary for renal and intestinal calcium (re)absorption.American journal of physiology. Renal physiology · 2012Article
- Bone disease in pediatric idiopathic hypercalciuria.World journal of nephrology · 2012Review
- Determinants of osteopenia in male renal-stone-disease patients with idiopathic hypercalciuria.Clinical journal of the American Society of Nephrology : CJASN · 2011Article
- Bone disease in medullary sponge kidney and effect of potassium citrate treatment.Clinical journal of the American Society of Nephrology : CJASN · 2009Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
Abstract
Observational and epidemiologic studies alike have shown that idiopathic hypercalciuric (IH) stone-forming patients typically show bone mineral density scores that are significantly lower than those observed for age- and sex-matched normal subjects or those for nonhypercalciuric stone-forming patients. Most of these studies have relied on changes in bone mineral density and have not explored the mechanism(s) involved. There have been a small number of studies that have relied on dynamic bone histomorphometry to ascertain the nature of the bone defect in IH patients. When performed, these studies clearly have shown increased bone resorption and high bone turnover in patients with fasting hypercalciuria whereas suppressed bone formation indices are the most consistent finding in patients with the absorptive variant of IH. The causes of this apparent difference in bone remodeling between the 2 variants of IH still is uncertain. Available evidence suggests that potential mechanisms may be dependent in large part to genetic, metabolic, and nutritional causes of hypercalciuria and bone loss in patients with IH.
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Registered trials
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