Are podocytes passive or provocative in proteinuric glomerular pathology?

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

9 citing papers in PubMed.

1. Role of baseline soluble tumor necrosis factor receptor 2 as a biomarker in primary podocytopathy: Implications for renal impairment and disease progression.BMC nephrology · 2024
   Article
2. New insights into the immune functions of podocytes: the role of complement.Molecular and cellular pediatrics · 2023
   Review
3. Valproate hampers podocyte acquisition of immune phenotypes via intercepting the GSK3β facilitated NFkB activation.Oncotarget · 2017
   Article
4. The level of urinary semaphorin3A is associated with disease activity in patients with minimal change nephrotic syndrome.International journal of nephrology and renovascular disease · 2017
   Article
5. Unfractionated bone marrow cells attenuate paraquat-induced glomerular injury and acute renal failure by modulating the inflammatory response.Scientific reports · 2016
   Article
6. Crosstalk between protective autophagy and NF-κB signal in high glucose-induced podocytes.Molecular and cellular biochemistry · 2014
   Article
7. LPS and PAN-induced podocyte injury in an in vitro model of minimal change disease: changes in TLR profile.Journal of cell communication and signaling · 2013
   Article
8. TNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α.Laboratory investigation; a journal of technical methods and pathology · 2011
   Article
9. alpha- and beta-Adducin polymorphisms affect podocyte proteins and proteinuria in rodents and decline of renal function in human IgA nephropathy.Journal of molecular medicine (Berlin, Germany) · 2010
   Article

Corrections and comments

• Comment on
  TLR4 links podocytes with the innate immune system to mediate glomerular injury.2008

Authors and funding

1 author.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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