ReviewSeminars in immunopathology2007
Anti-neutrophil cytoplasmic (ANCA) and anti-glomerular basement membrane (GBM) autoantibodies in necrotizing and crescentic glomerulonephritis.
Review in Seminars in immunopathology, 2007. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.
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Who cites it
14 citing papers in PubMed, 50 citations in OpenAlex.
- Nephrotic syndrome due to minimal-change disease superimposed on anti-glomerular basement membrane antibody positive glomerulonephritis; a case report.BMC nephrology · 2020Article
- Inhibitory Anti-Peroxidasin Antibodies in Pulmonary-Renal Syndromes.Journal of the American Society of Nephrology : JASN · 2018Article
- Crescentic glomerulonephritis with dual positive anti-GBM and C-ANCA/PR3 antibodies.Clinical nephrology. Case studies · 2016Article
- Nodular glomerulosclerosis with anti-glomerular basement membrane-like glomerulonephritis; a distinct pattern of kidney injury observed in smokers.Clinical kidney journal · 2014Article
- Lutheran/basal cell adhesion molecule accelerates progression of crescentic glomerulonephritis in mice.Kidney international · 2014Article
- Pulmonary renal syndrome in a child with coexistence of anti-neutrophil cytoplasmic antibodies and anti-glomerular basement membrane disease: case report and literature review.BMC nephrology · 2013Review
- Coexistence of ANCA-associated glomerulonephritis and anti-phospholipase A(2) receptor antibody-positive membranous nephropathy.Clinical kidney journal · 2012Article
- Crescentic Glomerulonephritis with Anti-GBM and p-ANCA Antibodies.Case reports in nephrology · 2012Article
- Asymptomatic autoantibodies associate with future anti-glomerular basement membrane disease.Journal of the American Society of Nephrology : JASN · 2011Article
- Advances in human antiglomerular basement membrane disease.Nature reviews. Nephrology · 2011Review
- Anti-hLAMP2-antibodies and dual positivity for anti-GBM and MPO-ANCA in a patient with relapsing pulmonary-renal syndrome.BMC nephrology · 2011Article
- Microscopic polyangiitis.Rheumatic diseases clinics of North America · 2010Review
- Poly(ADP-ribose) polymerase-1 regulates the progression of autoimmune nephritis in males by inducing necrotic cell death and modulating inflammation.Journal of immunology (Baltimore, Md. : 1950) · 2009Article
- Article
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Authors and funding
3 authors at 3 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Anti-neutrophil cytoplasmic autoantibodies (ANCA) and anti-glomerular basement membrane (GBM) necrotizing and crescentic glomerulonephritis are aggressive and destructive glomerular diseases that are associated with and probably caused by circulating ANCA and anti-GBM antibodies. These necrotizing lesions are manifested by acute nephritis and deteriorating kidney function often accompanied by distinctive clinical features of systemic disease. Prompt diagnosis requires clinical acumen that allows for the prompt institution of therapy aimed at removing circulating autoantibodies and quelling the inflammatory process. Continuing exploration of the etiology and pathogenesis of these aggressive inflammatory diseases have gradually uncovered new paradigms for the cause of and more specific therapy for these particular glomerular disorders and for autoimmune glomerular diseases in general.
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