ReviewInternational journal of immunogenetics2007
Genetic defects in common variable immunodeficiency.
Review in International journal of immunogenetics, 2007. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
11 citing papers in PubMed, 31 citations in OpenAlex.
- Whole exome sequencing identifies compound heterozygous variants ofSAGE open medicine · 2020Article
- Killer-cell immunoglobulin-like receptor and human leukocyte antigen-C genes in common variable immunodeficiency.Wiener klinische Wochenschrift · 2016Article
- Genetic defects and the role of helper T-cells in the pathogenesis of common variable immunodeficiency.Advanced biomedical research · 2014Review
- Three different classifications, B lymphocyte subpopulations, TNFRSF13B (TACI), TNFRSF13C (BAFF-R), TNFSF13 (APRIL) gene mutations, CTLA-4 and ICOS gene polymorphisms in Turkish patients with common variable immunodeficiency.Journal of clinical immunology · 2012Article
- B cell responses to CpG correlate with CXCL16 expression levels in common variable immunodeficiency.TheScientificWorldJournal · 2012Article
- Altered spectrum of somatic hypermutation in common variable immunodeficiency disease characteristic of defective repair of mutations.Immunogenetics · 2011Article
- Analysis of TACI mutations in CVID & RESPI patients who have inherited HLA B*44 or HLA*B8.BMC medical genetics · 2009Article
- Common variable immunodeficiency in horses is characterized by B cell depletion in primary and secondary lymphoid tissues.Journal of clinical immunology · 2009Article
- Genetic polymorphism study of regulatory B cell molecules and cellular immunity function in an adult patient with Common Variable Immunodeficiency.Hippokratia · 2008Article
- Homologous recombination is necessary for normal lymphocyte development.Molecular and cellular biology · 2008Article
- Familial inheritance and screening of first-degree relatives in common variable immunodeficiency and immunoglobulin A deficiency patients.International journal of immunopathology and pharmacologyArticle
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Common variable immunodeficiency (CVID) is the most frequent clinically manifested primary immunodeficiency. According to clinical and laboratory findings, CVID is a heterogeneous group of diseases. Recently, the defects of molecules regulating activation and terminal differentiation of B lymphocytes have been described in some patients with CVID. In this study, we show the overview of deficiencies of inducible costimulator, transmembrane activator and calcium-modulator and cytophilin ligand interactor, CD19 molecules, their genetic basis, pathogenesis and clinical manifestations.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.