ArticleVision research2007
Storage of an oculomotor motion aftereffect.
Article in Vision research, 2007. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
What it found
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Who cites it
8 citing papers in PubMed, 19 citations in OpenAlex.
- Afternystagmus in darkness after suppression of optokinetic nystagmus: an interaction of motion aftereffect and retinal afterimages.Experimental brain research · 2014Trial
- Vertical optokinetic eye movements in the larval zebrafish.The Journal of experimental biology · 2026Article
- Visual Snow Is Susceptible to the Motion Aftereffect.Investigative ophthalmology & visual science · 2025Article
- Dissociating representations of affect and motion in visual cortices.Cognitive, affective & behavioral neuroscience · 2023Article
- Sustained Rhythmic Brain Activity Underlies Visual Motion Perception in Zebrafish.Cell reports · 2016Article
- Emotional Actions Are Coded via Two Mechanisms: With and without Identity Representation.Frontiers in psychology · 2016Article
- The effects of prolonged viewing of motion on short-latency ocular following responses.Experimental brain research · 2009Article
- The influence of motion signals in hand movements.Experimental brain research · 2008Article
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Authors and funding
2 authors at 2 institutions in 1 country.
Funding
Abstract
Adaptation to motion produces a motion aftereffect (MAE), where illusory, oppositely-directed motion is perceived when viewing a stationary image. A common hypothesis for motion adaptation is that it reflects an imbalance of activity caused by neuronal fatigue. However, the perceptual MAE exhibits storage, in that the MAE appears even after a prolonged period of darkness is interposed between the adapting stimulus and the test, suggesting that fatigue cannot explain the perceptual MAE. We asked whether neural fatigue was a viable explanation for the oculomotor MAE (OMAE) by testing if the OMAE exhibits storage. Human observers were adapted with moving, random-dot cinematograms. Following adaptation, they generated an oculomotor MAE (OMAE), with both pursuit and saccadic components. The OMAE occurred in the presence of a visual test stimulus, but not in the dark. When the test stimulus was introduced after the dark period, the OMAE reappeared, analogous to perceptual MAE storage. The results suggest that fatigue cannot explain the OMAE, and that visual stimulation is necessary to elicit it. We propose a model in which adaptation recalibrates the motion-processing network by adjusting the weights of the inputs to neurons in the middle-temporal (MT) area.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.