ArticleThe American journal of pathology1991
Enhanced expression of neural cell adhesion molecules and tenascin (cytotactin) during wound healing.
Article in The American journal of pathology, 1991. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 32 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
32 citing papers in PubMed, 110 citations in OpenAlex.
- Roles of K-channel activity in feather bud morphogenesis.Developmental biology · 2026Article
- Conserved regulatory switches for the transition from natal down to juvenile feather in birds.Nature communications · 2024Article
- Transition from natal downs to juvenile feathers: conserved regulatory switches in Neoaves.Research square · 2023Article
- Injectable Cell-Laden Nanofibrous Matrix for Treating Annulus Fibrosus Defects in Porcine Model: An Organ Culture Study.Life (Basel, Switzerland) · 2022Article
- Research status and hot topics of the effects of skin innervation on wound healing from 1959 to 2022: A bibliometric analysis.Frontiers in surgery · 2022Article
- Cyclic growth of dermal papilla and regeneration of follicular mesenchymal components during feather cycling.Development (Cambridge, England) · 2021Article
- The Effects of Premature Tooth Extraction and Damage on Replacement Timing in the Green Iguana.Integrative and comparative biology · 2020Article
- Multiple Regulatory Modules Are Required for Scale-to-Feather Conversion.Molecular biology and evolution · 2018Article
- Article
- Regeneration of reptilian scales after wounding: neogenesis, regional difference, and molecular modules.Regeneration (Oxford, England) · 2014Article
- Cellular distribution and gene expression profile during flexor tendon graft repair: A novel tissue engineering approach(*).Journal of tissue engineering · 2013Article
- Lack of CXC chemokine receptor 3 signaling leads to hypertrophic and hypercellular scarring.The American journal of pathology · 2010Article
- Transforming growth factors beta coordinate cartilage and tendon differentiation in the developing limb mesenchyme.The Journal of biological chemistry · 2009Article
- Role of Annexin-II in GI cancers: interaction with gastrins/progastrins.Cancer letters · 2007Review
- Chondroitin sulphate proteoglycans: preventing plasticity or protecting the CNS?Journal of anatomy · 2004Review
- Tenascin expression in primary and recurrent breast carcinomas and the effect of tenascin on breast tumor cell cultures.Pathology oncology research : POR · 2000Article
- Mechanical forces induce scar remodeling. Study in non-pressure-treated versus pressure-treated hypertrophic scars.The American journal of pathology · 1999Article
- Expression of SPARC (secreted protein, acidic and rich in cysteine) in healing intestinal anastomoses and short bowel syndrome in rats.Digestive diseases and sciences · 1999Article
- Differential expression of tenascin in the skin during hapten-induced dermatitis.Histochemistry and cell biology · 1996Article
- Tenascin-C and human tendon degeneration.The American journal of pathology · 1996Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors at 1 institution in 1 country.
Funding
Abstract
Both neural cell adhesion molecules (N-CAM) and tenascin (cytotactin) are important in embryonic morphogenesis but their expression is reduced greatly in adults. This study examined whether they are induced during wound healing. The spatial and temporal expression patterns of these two and other adhesion molecules in the healing of skin, cartilage, and tendon were compared. Neural cell adhesion molecules, tenascin, and fibronectin are induced in the granulation tissue but the order of prevalence is fibronectin, tenascin, N-CAM. The order of appearance is N-CAM and fibronectin, and then tenascin. The order of disappearance is N-CAM, tenascin, fibronectin. Liver cell adhesion molecules are present in the epidermis undergoing re-epithelization. Explant cultures showed that N-CAM and tenascin are synthesized by wound fibroblasts. These results suggest that N-CAM and tenascin, widely used in embryonic morphogenesis, are induced in a variety of connective tissues during wound healing.
Indexed as
Identifiers
1704192PMC1886190W1883473411What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.