Evidence map›Paper›PMID 17028369›Full record

ReviewNeuromolecular medicine2006

The genetics of autism spectrum disorders.

Dorothy E Grice, Joseph D Buxbaum

Abstract readReview
PubMed Publisher
In one paragraph

Review in Neuromolecular medicine, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed.

  1. Alpha-neurexins in health and disease.Frontiers in molecular neuroscience · 2025
    Review
  2. Autism-like symptoms by exposure to air pollution and valproic acid-induced in male rats.Environmental science and pollution research international · 2022
    Article
  3. Oxidative stress indices in ASD children in Sub-Sahara Africa.Journal of neurodevelopmental disorders · 2021
    Article
  4. Article
  5. Article
  6. Effects of PMParticle and fibre toxicology · 2020
    Article
  7. Review
  8. Review
  9. Article
  10. Review
  11. Article
  12. Environmental risk factors for autism.Emerging health threats journal · 2011
    Article
  13. Article
  14. Article
  15. Article
  16. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Dorothy E GriceDepartment of Psychiatry, UMDNJ/New Jersey Medical School, Newark, NJ 07103, USA.
Joseph D Buxbaum

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Epidemiological twin studies demonstrate that autism spectrum disorders (ASDs) represent genetic disorders. Subsequent analyses indicate that the causes of ASDs include less common single-gene mutations and chromosomal abnormalities, as well as ASDs caused by multiple interacting genes of weak effect. Genome-wide linkage analysis has identified several susceptibility loci for the ASDs, and positional and functional candidate genes have been identified that appear to represent susceptibility genes for the ASDs. Analysis of additional larger samples and the use of genome-wide association and high-throughput variant detection will lead to the identification of further genes for ASDs.

Indexed as

Genetic Predisposition to DiseaseGenetic VariationGenome, HumanAutistic DisorderChromosome AberrationsChromosome MappingGenetic TestingHumansTwin Studies as Topic

Identifiers

PMID17028369

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.