ReviewCritical care (London, England)2006
Hyperinsulinemia-euglycemia therapy: a useful tool in treating calcium channel blocker poisoning.
Review in Critical care (London, England), 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
6 citing papers in PubMed.
- Before and After Study of Pharmacists' and Students' Knowledge of Two Novel Antidotes: High-Dose Insulin Euglycemia and Intravenous Fatty Acid Emulsion 20.Hospital pharmacy · 2015Article
- High dose insulin therapy, an evidence based approach to beta blocker/calcium channel blocker toxicity.Daru : journal of Faculty of Pharmacy, Tehran University of Medical Sciences · 2014Article
- Common causes of poisoning: etiology, diagnosis and treatment.Deutsches Arzteblatt international · 2013Review
- Hyperinsulinemia euglycemia therapy for calcium channel blocker overdose: a case report.Texas Heart Institute journal · 2012Article
- Prolonged Refractory Hypotension following Combined Amlodipine and Losartan Ingestion Responsive to Metaraminol.Case reports in medicine · 2011Article
- Insulin-dependent rescue from cardiogenic shock is not mediated by phospholamban phosphorylation.Clinical toxicology (Philadelphia, Pa.) · 2009Article
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2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Hyperinsulinemia-euglycemia (HIE) therapy, when initiated promptly and aggressively, may offer considerable advantages in the treatment of calcium channel blocker poisoning. Although its mechanism of action is uncertain, HIE improves the efficiency with which the poisoned myocardium uses metabolic fuel, the end result of which is improvements in inotropy and other cardiovascular parameters. Although HIE is not universally accepted, the reports included in the previous issue of Critical Care should prompt clinicians to consider HIE an appropriate therapy specifically for calcium channel blocker poisoning.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.