Evidence map›Paper›PMID 16815214›Full record

ReviewNeuroRx : the journal of the American Society for Experimental NeuroTherapeutics2006

Single cell gene expression profiling in Alzheimer's disease.

Stephen D Ginsberg, Shaoli Che, Scott E Counts, Elliott J Mufson

Open access · bronzeAbstract readReview
In one paragraph

Review in NeuroRx : the journal of the American Society for Experimental NeuroTherapeutics, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 48 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
48citing papers in PubMed, 1 pooled it
4.1field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

48 citing papers in PubMed, 1 synthesis or guideline pooled it, 75 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 4 institutions in 1 country.

Stephen D GinsbergCenter for Dementia Research, Nathan Kline Institute, Orangeburg, NY 10962, USA. ginsberg@nki.rfmh.org
Shaoli Che
Scott E Counts
Elliott J Mufson
Nathan Kline Institute for Psychiatric Research · USNew York University · USRush University Medical Center · USUniversity of Illinois Chicago · US

Funding

SUPPLEMENT TO RUSH ALZHEIMERS DISEASE CENTER COREP30AG010161 · NIA · RUSH UNIVERSITY MEDICAL CENTER · PI BENNETT, DAVID ALAN · 1991 to 2020
$49.1M
Uncovering Alzheimer's disease risk mechanisms through neuron-specific analysis of autophagy and endosomal-lysosomal functionP01AG017617 · NIA · NATHAN S. KLINE INSTITUTE FOR PSYCH RES · PI NIXON, RALPH A. · 2000 to 2021
$40.0M
TAU, AB, SYNUCLEIN AND NITRATIVE/OXIDATIVE DAMAGE IN MCIP01AG014449 · NIA · UNIVERSITY OF PITTSBURGH · PI IKONOMOVIC, MILOS D · 1997 to 2024
$39.7M
Tau Truncation & Conformation In Alzheimer's Disease ProgressionP01AG009466 · NIA · RUSH UNIVERSITY MEDICAL CENTER · PI BINDER, LESTER IRVIN · 1991 to 2012
$14.3M
Single Cell Gene Expression Profiling in hTau MiceP01NS048447 · NINDS · NEW YORK STATE PSYCHIATRIC INSTITUTE DBA RESEARCH FOUNDATION FOR MENTAL HYGIENE, INC · PI DUFF, KAREN · 2005 to 2009
$6.4M
GALANIN IN ALZHEIMER'S DISEASER01AG010668 · NIA · RUSH UNIVERSITY MEDICAL CENTER · PI MUFSON, ELLIOTT JAY · 1991 to 2011
$4.1M
Cellular and Molecular Medial Temporal Lobe Pathology in Elderly PreMCI subjectsR01AG043375 · NIA · ST. JOSEPH'S HOSPITAL AND MEDICAL CENTER · PI GINSBERG, STEPHEN D, MUFSON, ELLIOTT JAY · 2013 to 2017
$3.6M
Lesion Induced Synaptic PlasticityR01NS043939 · NINDS · NATHAN S. KLINE INSTITUTE FOR PSYCH RES · PI GINSBERG, STEPHEN D. · 2001 to 2004
$1.3M
Proteomics of mild cognitive impairment in the elderlyR21AG026032 · NIA · RUSH UNIVERSITY MEDICAL CENTER · PI COUNTS, SCOTT E · 2005 to 2006
$342k
Molecular Diagnosis Utilizing Cerebrospinal FluidR21CA094520 · NCI · NATHAN S. KLINE INSTITUTE FOR PSYCH RES · PI GINSBERG, STEPHEN D. · 2002 to 2003
$189k
NIA NIH HHS AG09466NIA NIH HHS AG10161NIA NIH HHS AG10688NIA NIH HHS AG14449NIA NIH HHS AG17617NIA NIH HHS AG26032NIA NIH HHS P01 AG009466NIA NIH HHS P01 AG014449NIA NIH HHS P01 AG017617NIA NIH HHS R01 AG010668NIA NIH HHS R01 AG043375NINDS NIH HHS NS43939NINDS NIH HHS NS48447NINDS NIH HHS P01 NS048447NINDS NIH HHS R01 NS043939
6 · The paper itself

Abstract

Development and implementation of microarray techniques to quantify expression levels of dozens to hundreds to thousands of transcripts simultaneously within select tissue samples from normal control subjects and neurodegenerative diseased brains has enabled scientists to create molecular fingerprints of vulnerable neuronal populations in Alzheimer's disease (AD) and related disorders. A goal is to sample gene expression from homogeneous cell types within a defined region without potential contamination by expression profiles of adjacent neuronal subpopulations and nonneuronal cells. The precise resolution afforded by single cell and population cell RNA analysis in combination with microarrays and real-time quantitative polymerase chain reaction (qPCR)-based analyses allows for relative gene expression level comparisons across cell types under different experimental conditions and disease progression. The ability to analyze single cells is an important distinction from global and regional assessments of mRNA expression and can be applied to optimally prepared tissues from animal models of neurodegeneration as well as postmortem human brain tissues. Gene expression analysis in postmortem AD brain regions including the hippocampal formation and neocortex reveals selectively vulnerable cell types share putative pathogenetic alterations in common classes of transcripts, for example, markers of glutamatergic neurotransmission, synaptic-related markers, protein phosphatases and kinases, and neurotrophins/neurotrophin receptors. Expression profiles of vulnerable regions and neurons may reveal important clues toward the understanding of the molecular pathogenesis of various neurological diseases and aid in identifying rational targets toward pharmacotherapeutic interventions for progressive, late-onset neurodegenerative disorders such as mild cognitive impairment (MCI) and AD.

Indexed as

Alzheimer DiseaseAmyloid beta-Protein PrecursorAnimalsBrainGene ExpressionGene Expression ProfilingHumansMembrane ProteinsMicroarray AnalysisMutationNeuronsPresenilin-1Presenilin-2tau ProteinsAmyloid beta-Protein PrecursorMembrane ProteinsPresenilin-1Presenilin-2PSEN1 protein, humanPSEN2 protein, humantau Proteins

Identifiers

PMID16815214
PMCPMC3593387
OpenAlexW1979242019

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.