Evidence map›Paper›PMID 16806461›Full record

ReviewCell calcium2006

TRP channels and Ca2+ signaling.

Baruch Minke

Open access · greenAbstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Cell calcium, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 66 papers.

0numbers the graph read from it
0cells of the map it votes in
66citing papers in PubMed
4.5field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

66 citing papers in PubMed, 154 citations in OpenAlex.

  1. Review
  2. Review
  3. Review
  4. The Role of TRPM7 in Oncogenesis.International journal of molecular sciences · 2024
    Review
  5. Review
  6. Review
  7. Review
  8. Review
  9. Article
  10. Review
  11. Thioredoxin-dependent system. Application of inhibitors.Journal of enzyme inhibition and medicinal chemistry · 2021
    Review
  12. Review
  13. Review
  14. Article
  15. Role of TRP Channels in Shaping the Gut Microbiome.Pathogens (Basel, Switzerland) · 2020
    Article
  16. Review
  17. Review
  18. TRPV6 as A Target for Cancer Therapy.Journal of Cancer · 2020
    Review
  19. Review
  20. Article

6 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Baruch MinkeDepartment of Physiology and the Kühne Minerva Center for Studies of Visual Transduction, The Hebrew University of Jerusalem, Jerusalem 91120, Israel. minke@md.huji.ac.il
Hebrew University of Jerusalem · IL

Funding

GENETIC DISSECTION OF PHOTOTRANSDUCTIONR01EY003529 · NEI · HEBREW UNIVERSITY OF JERUSALEM · PI MINKE, BARUCH · 1985 to 2011
$1.8M
NEI NIH HHS EY 03529NEI NIH HHS R01 EY003529
6 · The paper itself

Abstract

There is a rapidly growing interest in the family of transient receptor potential (TRP) channels because TRP channels are not only important for many sensory systems, but they are crucial components of the function of neurons, epithelial, blood and smooth muscle cells. These facts make TRP channels important targets for treatment of diseases arising from the malfunction of these channels in the above cells and for treatment of inflammatory pain. TRP channels are also important for a growing number of genetic diseases arising from mutations in various types of TRP channels. The Minerva-Gentner Symposium on TRP channels and Ca(2+) signaling, which took place in Eilat, Israel (February 24-28, 2006) has clearly demonstrated that the study of TRP channels is a newly emerging field of biomedicine with prime importance. In the Eilat symposium, investigators who have contributed seminal publications and insight into the TRP field presented their most recent, and in many cases still unpublished, studies. The excellent presentations and excitement generated by them demonstrated that much progress has been achieved. Nevertheless, it was also evident that the field of TRP channels is still in its infancy in comparison to other fields of ion channels, and even the fundamental knowledge of the gating mechanism of TRP channels is still unsolved. The beautiful location of the symposium, together with informal intensive discussions among the participants, contributed to the success of this meeting.

Indexed as

Calcium SignalingAnimalsDrosophila ProteinsHumansTransient Receptor Potential ChannelsTRPC Cation ChannelsTRPM Cation ChannelsTRPV Cation ChannelsDrosophila ProteinsMCOLN1 protein, humanTransient Receptor Potential ChannelsTRPC Cation ChannelsTRPM Cation Channelstrp protein, DrosophilaTRPV Cation Channels

Identifiers

PMID16806461
PMCPMC1934411
OpenAlexW2053095154

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.