ReviewCellular microbiology2006
Hosting the severe acute respiratory syndrome coronavirus: specific cell factors required for infection.
Review in Cellular microbiology, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
15 citing papers in PubMed.
- Why Should Natural Principles Be Simple?Philosophia (Ramat-Gan, Israel) · 2022Article
- Advances in the design and development of SARS-CoV-2 vaccines.Military Medical Research · 2021Review
- SARS-CoV-2: An Overview of Virus Genetics, Transmission, and Immunopathogenesis.International journal of environmental research and public health · 2021Review
- Short linear motif candidates in the cell entry system used by SARS-CoV-2 and their potential therapeutic implications.Science signaling · 2021Review
- Feline coronavirus replication is affected by both cyclophilin A and cyclophilin B.The Journal of general virology · 2017Article
- Cellular peptidyl-prolyl cis/trans isomerase Pin1 facilitates replication of feline coronavirus.Antiviral research · 2016Article
- Article
- Human coronavirus NL63 replication is cyclophilin A-dependent and inhibited by non-immunosuppressive cyclosporine A-derivatives including Alisporivir.Virus research · 2014Article
- Altering SARS coronavirus frameshift efficiency affects genomic and subgenomic RNA production.Viruses · 2013Article
- Replication of human coronaviruses SARS-CoV, HCoV-NL63 and HCoV-229E is inhibited by the drug FK506.Virus research · 2012Article
- Cyclosporin A inhibits the replication of diverse coronaviruses.The Journal of general virology · 2011Article
- Unconventional use of LC3 by coronaviruses through the alleged subversion of the ERAD tuning pathway.Viruses · 2011Review
- Qualitative and quantitative ultrastructural analysis of the membrane rearrangements induced by coronavirus.Cellular microbiology · 2010Article
- The role of programmed-1 ribosomal frameshifting in coronavirus propagation.Frontiers in bioscience : a journal and virtual library · 2008Review
- Synthesis and biological evaluation of nucleoside analogues having 6-chloropurine as anti-SARS-CoV agents.Bioorganic & medicinal chemistry letters · 2007Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
As with all viruses, the severe acute respiratory syndrome coronavirus (SARS-CoV) utilizes specific host cell factors during its infection cycle. Some of these factors have been identified and are now increasingly scrutinized as targets to intervene with infection. In this brief review, we describe the current understanding of how the SARS-CoV is able to use the cellular machinery for its replication.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.