Evidence map›Paper›PMID 1663159›Full record

ArticleThe Journal of physiology1991

Dependence of cytosolic calcium in differentiating rat pheochromocytoma cells on calcium channels and intracellular stores.

B F Reber, H Reuter

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of physiology, 1991. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
3.3field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 68 citations in OpenAlex.

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  4. Nerve growth factor-induced differentiation changes the cellular organization of regulated Peptide release by PC12 cells.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2002
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

B F ReberDepartment of Pharmacology, University of Bern, Switzerland.
H Reuter
University of Bern · CH

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

1. The rat clonal pheochromocytoma cell line (PC12) was used to study changes in the free intracellular Ca2+ concentration [( Ca2+]i) that are related to the distribution of L-type (dihydropyridine-sensitive) and N-type (omega-conotoxin-sensitive) calcium channels during nerve growth factor (NGF)-induced outgrowth of neurites. Changes in [Ca2+]i during K+ depolarization were recorded by means of Fura-2 single-cell microfluorimetry. 2. The basal [Ca2+]i of cells at rest was not altered by long-term treatment with NGF, neither in the cell bodies nor in the growth cones. K+ depolarization of the cells caused a rise in [Ca2+]i. 3. The dihydropyridine (DHP) nifedipine alone, or together with omega-conotoxin (omega-CgTX), were similarly effective in inhibiting the K(+)-induced increase in [Ca2+]i in untreated and NGF-treated cell bodies, arguing for a preferential distribution of L-type Ca2+ channels in this cell area. By contrast, after 6-7 days exposure to NGF the K(+)-induced initial transient rise of [Ca2+]i in growth cones was very sensitive to omega-CgTX, whereas nifedipine affected only the sustained rise. 4. PC12 cells also contain caffeine- and inositol trisphosphate (IP3)-sensitive intracellular Ca2+ stores. Addition of 30 mM-caffeine caused a fast transient rise in [Ca2+]i. The extent of filling of the caffeine-sensitive pool affected basal [Ca2+]i. These Ca2+ storage sites were empty under normal culture conditions. However, a single K+ depolarization caused filling of the stores, followed by spontaneous depletion (50% in about 5 min) after wash-out of high [K+]o. When the caffeine-sensitive stores were empty, the rise in [Ca2+]i was attenuated during submaximal depolarization. Caffeine-sensitive Ca2+ stores were also present in some growth cones, though with much smaller capacities than in cell bodies. 5. Mobilization of Ca2+ from the IP3-sensitive store, by bradykinin exposure, was found to be independent of the caffeine-sensitive pool. There was no apparent 'cross-talk' between both Ca2+ pools. 6. We conclude that changes in [Ca2+]i in cell bodies depend on both membrane Ca2+ channels and intracellular Ca2+ stores. During NGF-induced differentiation there is a predominance of N-type Ca2+ channels in growth cones, while Ca2+ stores are of minor importance in these structures.

Indexed as

AnimalsCaffeineCalciumCalcium Channel BlockersCalcium ChannelsCell DifferentiationCytosolPheochromocytomaPotassiumRatsTumor Cells, CulturedCaffeineCalciumCalcium Channel BlockersCalcium ChannelsPotassium

Identifiers

PMID1663159
PMCPMC1181454
OpenAlexW2028553251

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.