Evidence map›Paper›PMID 16570343›Full record

ReviewWorld journal of gastroenterology2006

Pathogenesis of columnar-lined esophagus.

Kamal E Bani-Hani, Bayan K Bani-Hani

Abstract readReview
In one paragraph

Review in World journal of gastroenterology, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Cardiac mucosa indicates risk for Barrett esophagus.World journal of gastroenterology · 2006
    Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors.

Kamal E Bani-HaniDepartment of Surgery, Faculty of Medicine, Jordan University of Science and Technology, PO Box 3030, Irbid 22110, Jordan. banihani60@yahoo.com
Bayan K Bani-Hani

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Since its initial description, the pathogenesis of the columnar-lined esophagus (CLE) has been surrounded by many controversies. The first controversy is related to the existence of the condition itself. The second controversy centers on whether the CLE is a congenital or an acquired condition. In this article, we review the congenital and acquired theories of development of CLE and discuss the various factors in acquisition of CLE. The bulk of evidence in the literature suggests that CLE is an acquired condition.

Indexed as

AdenocarcinomaBarrett EsophagusEpitheliumEsophageal NeoplasmsEsophagusGastroesophageal RefluxHumansMucous MembranePrecancerous Conditions

Identifiers

PMID16570343
PMCPMC4124283

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.